Nociceptors as chronic drivers of pain and hyperreflexia after spinal cord injury: an adaptive-maladaptive hyperfunctional state hypothesis.
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Use of von Frey filaments to assess nociceptive sensitization in the hornworm, Manduca sextaIn Vivo Imaging of Human Neuroinflammation.Squid have nociceptors that display widespread long-term sensitization and spontaneous activity after bodily injuryDysregulation of Kv3.4 channels in dorsal root ganglia following spinal cord injuryThe Transcriptional Response of Neurotrophins and Their Tyrosine Kinase Receptors in Lumbar Sensorimotor Circuits to Spinal Cord Contusion is Affected by Injury Severity and Survival Time.Persistent Electrical Activity in Primary Nociceptors after Spinal Cord Injury Is Maintained by Scaffolded Adenylyl Cyclase and Protein Kinase A and Is Associated with Altered Adenylyl Cyclase Regulation.Delayed Exercise Is Ineffective at Reversing Aberrant Nociceptive Afferent Plasticity or Neuropathic Pain After Spinal Cord Injury in Rats.Calcineurin Dysregulation Underlies Spinal Cord Injury-Induced K+ Channel Dysfunction in DRG Neurons.Persistent pain after spinal cord injury is maintained by primary afferent activity.Challenges and opportunities of sensory plasticity after SCI.Origins of Phantom Limb Pain.How is chronic pain related to sympathetic dysfunction and autonomic dysreflexia following spinal cord injury?Dynamic "Range of Motion" Hindlimb Stretching Disrupts Locomotor Function in Rats with Moderate Subacute Spinal Cord Injuries.Activation of KCNQ Channels Suppresses Spontaneous Activity in Dorsal Root Ganglion Neurons and Reduces Chronic Pain after Spinal Cord Injury.Dorsal root ganglion neurons become hyperexcitable and increase expression of voltage-gated T-type calcium channels (Cav3.2) in paclitaxel-induced peripheral neuropathy.Epigenetic Modifications Associated to Neuroinflammation and Neuropathic Pain After Neural Trauma.Nociceptive Biology of Molluscs and Arthropods: Evolutionary Clues About Functions and Mechanisms Potentially Related to Pain
P2860
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P2860
Nociceptors as chronic drivers of pain and hyperreflexia after spinal cord injury: an adaptive-maladaptive hyperfunctional state hypothesis.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Nociceptors as chronic drivers ...... erfunctional state hypothesis.
@en
Nociceptors as chronic drivers ...... erfunctional state hypothesis.
@nl
type
label
Nociceptors as chronic drivers ...... erfunctional state hypothesis.
@en
Nociceptors as chronic drivers ...... erfunctional state hypothesis.
@nl
prefLabel
Nociceptors as chronic drivers ...... erfunctional state hypothesis.
@en
Nociceptors as chronic drivers ...... erfunctional state hypothesis.
@nl
P2860
P356
P1476
Nociceptors as chronic drivers ...... erfunctional state hypothesis.
@en
P2860
P356
10.3389/FPHYS.2012.00309
P577
2012-08-02T00:00:00Z