CD19-independent instruction of murine marginal zone B-cell development by constitutive Notch2 signaling.
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Akt and mTOR in B Cell Activation and DifferentiationApoptotic cell responses in the splenic marginal zone: a paradigm for immunologic reactions to apoptotic antigens with implications for autoimmunityMarginal zone B cells: virtues of innate-like antibody-producing lymphocytes.ADAM10 regulates transcription factor expression required for plasma cell functionThe intracellular domains of Notch1 and Notch2 are functionally equivalent during development and carcinogenesisBruton's Tyrosine Kinase Synergizes with Notch2 To Govern Marginal Zone B Cells in Nonobese Diabetic Mice.The coding genome of splenic marginal zone lymphoma: activation of NOTCH2 and other pathways regulating marginal zone development.Notch Signaling and the Skeleton.Notch-regulated periphery B cell differentiation involves suppression of E protein function.Requirement for Rictor in homeostasis and function of mature B lymphoid cells.IRF4 controls the positioning of mature B cells in the lymphoid microenvironments by regulating NOTCH2 expression and activity.Endothelial Plasmalemma Vesicle-Associated Protein Regulates the Homeostasis of Splenic Immature B Cells and B-1 B Cells.The genetic landscape of dural marginal zone lymphomas.Genetic landscape and deregulated pathways in B-cell lymphoid malignancies.Hajdu-Cheney Syndrome, a Disease Associated with NOTCH2 Mutations.CD19 regulates ADAM28-mediated Notch2 cleavage to control the differentiation of marginal zone precursors to MZ B cells.Hajdu Cheney Syndrome; report of a novel NOTCH2 mutation and treatment with denosumabTransitional B cells commit to marginal zone B cell fate by Taok3-mediated surface expression of ADAM10.Canonical Notch2 signaling determines biliary cell fates of embryonic hepatoblasts and adult hepatocytes independent of Hes1.The Hajdu Cheney Mutation Is a Determinant of B-Cell Allocation of the Splenic Marginal Zone.KLF2 mutation is the most frequent somatic change in splenic marginal zone lymphoma and identifies a subset with distinct genotype.Clinical and experimental aspects of notch receptor signaling: Hajdu-Cheney syndrome and related disorders.Notch2 activation ameliorates nephrosis.Induction of the Hajdu-Cheney Syndrome Mutation in CD19 B Cells in Mice Alters B-Cell Allocation but Not Skeletal Homeostasis.The splenic marginal zone shapes the phenotype of leukemia B cells and facilitates their niche-specific retention and survival.Whole-exome sequencing in splenic marginal zone lymphoma reveals mutations in genes involved in marginal zone differentiation.
P2860
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P2860
CD19-independent instruction of murine marginal zone B-cell development by constitutive Notch2 signaling.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
@zh-my
2011年学术文章
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2011年學術文章
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2011年學術文章
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2011年學術文章
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name
CD19-independent instruction o ...... constitutive Notch2 signaling.
@en
CD19-independent instruction o ...... constitutive Notch2 signaling.
@nl
type
label
CD19-independent instruction o ...... constitutive Notch2 signaling.
@en
CD19-independent instruction o ...... constitutive Notch2 signaling.
@nl
prefLabel
CD19-independent instruction o ...... constitutive Notch2 signaling.
@en
CD19-independent instruction o ...... constitutive Notch2 signaling.
@nl
P2093
P50
P1433
P1476
CD19-independent instruction o ...... constitutive Notch2 signaling
@en
P2093
Anne Draeseke
Brigitte Mack
Caroline Hojer
Franziska Hampel
Gabriele Marschall-Schröter
Lothar J Strobl
Marc Schmidt-Supprian
Stefanie Ehrenberg
Ursula Zimber-Strobl
P304
P356
10.1182/BLOOD-2010-12-325944
P407
P577
2011-07-27T00:00:00Z