about
The Trithorax group protein Ash2l and Saf-A are recruited to the inactive X chromosome at the onset of stable X inactivationXist and the order of silencingGenetic exploration of the exit from self-renewal using haploid embryonic stem cellsRing1B is crucial for the regulation of developmental control genes and PRC1 proteins but not X inactivation in embryonic cells.Haploid genomes illustrate epigenetic constraints and gene dosage effects in mammals.Derivation of haploid embryonic stem cells from mouse embryos.Establishment of epigenetic patterns in developmentThe histone deacetylase inhibitor sodium valproate causes limited transcriptional change in mouse embryonic stem cells but selectively overrides Polycomb-mediated Hoxb silencingMechanistic concepts in X inactivation underlying dosage compensation in mammals.SATB1 defines the developmental context for gene silencing by Xist in lymphoma and embryonic cells.Ring1B compacts chromatin structure and represses gene expression independent of histone ubiquitination.Polycomb complexes act redundantly to repress genomic repeats and genes.Histone acetylation and the maintenance of chromatin compaction by Polycomb repressive complexes.Establishment and Use of Mouse Haploid ES Cells.
P50
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P50
description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Martin Leeb
@ast
Martin Leeb
@en
Martin Leeb
@es
Martin Leeb
@nl
Martin Leeb
@sl
type
label
Martin Leeb
@ast
Martin Leeb
@en
Martin Leeb
@es
Martin Leeb
@nl
Martin Leeb
@sl
prefLabel
Martin Leeb
@ast
Martin Leeb
@en
Martin Leeb
@es
Martin Leeb
@nl
Martin Leeb
@sl
P1053
N-3861-2015
P106
P21
P31
P3829
P496
0000-0001-5114-4782