The Trithorax group protein Ash2l and Saf-A are recruited to the inactive X chromosome at the onset of stable X inactivation - Test2 - elhamkhani - TriplyDB

The Trithorax group protein Ash2l and Saf-A are recruited to the inactive X chromosome at the onset of stable X inactivation

The Trithorax group protein Ash2l and Saf-A are recruited to the inactive X chromosome at the onset of stable X inactivation

http://www.wikidata.org/entity/Q24632245

about

Function and evolution of the long noncoding RNA circuitry orchestrating X-chromosome inactivation in mammals The "lnc" between 3D chromatin structure and X chromosome inactivation X Inactivation Lessons from Differentiating Mouse Embryonic Stem Cells Long nonoding RNAs in the X-inactivation center Advances in understanding chromosome silencing by the long non-coding RNA Xist Wdr5 mediates self-renewal and reprogramming via the embryonic stem cell core transcriptional network A system for imaging the regulatory noncoding Xist RNA in living mouse embryonic stem cells.Active chromatin marks are retained on X chromosomes lacking gene or repeat silencing despite XIST/Xist expression in somatic cell hybrids.Regulation of Mammalian Gene Dosage by Long Noncoding RNAs.The Mbd1-Atf7ip-Setdb1 pathway contributes to the maintenance of X chromosome inactivation.The Xist lncRNA exploits three-dimensional genome architecture to spread across the X chromosome.Trithorax group proteins: switching genes on and keeping them active.Meta-analyses identify 13 loci associated with age at menopause and highlight DNA repair and immune pathways.Identification of Spen as a Crucial Factor for Xist Function through Forward Genetic Screening in Haploid Embryonic Stem Cells.Regulation of X-chromosome inactivation by the X-inactivation centre.Evaluating GWAS-identified SNPs for age at natural menopause among chinese women.Xist regulation and function explored.Evolutionary diversity and developmental regulation of X-chromosome inactivation.The inactive X chromosome adopts a unique three-dimensional conformation that is dependent on Xist RNA.Dosage compensation in mammals.Role of ATRX in chromatin structure and function: implications for chromosome instability and human disease Xist Exon 7 Contributes to the Stable Localization of Xist RNA on the Inactive X-Chromosome.Nuclear matrix factor hnRNP U/SAF-A exerts a global control of alternative splicing by regulating U2 snRNP maturation.Establishment of epigenetic patterns in development New and Xisting regulatory mechanisms of X chromosome inactivation Impact of flanking chromosomal sequences on localization and silencing by the human non-coding RNA XIST.Smchd1-dependent and -independent pathways determine developmental dynamics of CpG island methylation on the inactive X chromosome.Spreading of X chromosome inactivation via a hierarchy of defined Polycomb stations.Insight into lncRNA biology using hybridization capture analyses.Epigenetics of pluripotent cells.Noncoding RNA and Polycomb recruitment.Reactivation of the inactive X chromosome in development and reprogramming The evolutionary pathway of x chromosome inactivation in mammals eXIST with matrix-associated proteins.Mechanistic insights into chromosome-wide silencing in X inactivation.A scaffold for X chromosome inactivation.X chromosome inactivation: a silence that needs to be broken.Gracefully ageing at 50, X-chromosome inactivation becomes a paradigm for RNA and chromatin control.Biological and bioinformatical approaches to study crosstalk of long-non-coding RNAs and chromatin-modifying proteins.X-chromosome inactivation in development and cancer.

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The Trithorax group protein Ash2l and Saf-A are recruited to the inactive X chromosome at the onset of stable X inactivation

http://www.wikidata.org/entity/Q24632245

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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The Trithorax group protein As ...... onset of stable X inactivation

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The Trithorax group protein As ...... onset of stable X inactivation

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Anton Wutz

http://www.w3.org/2001/XMLSchema#string

Dieter Pullirsch

http://www.w3.org/2001/XMLSchema#string

Günter Steiner

http://www.w3.org/2001/XMLSchema#string

Hiroyuki Kishimoto

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Renate Härtel

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Leukemia proto-oncoprotein MLL forms a SET1-like histone methyltransferase complex with menin to regulate Hox gene expression

Human Sin3 deacetylase and trithorax-related Set1/Ash2 histone H3-K4 methyltransferase are tethered together selectively by the cell-proliferation factor HCF-1

The human trithorax protein hASH2 functions as an oncoprotein

Weak but uniform enrichment of the histone variant macroH2A1 along the inactive X chromosome

The novel SAR-binding domain of scaffold attachment factor A (SAF-A) is a target in apoptotic nuclear breakdown.

Genome-wide maps of chromatin state in pluripotent and lineage-committed cells

XIST RNA paints the inactive X chromosome at interphase: evidence for a novel RNA involved in nuclear/chromosome structure

A novel role for Xist RNA in the formation of a repressive nuclear compartment into which genes are recruited when silenced

Synergism of Xist RNA, DNA methylation, and histone hypoacetylation in maintaining X chromosome inactivation

A chromosomal memory triggered by Xist regulates histone methylation in X inactivation

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935-43

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scholarly article

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10.1242/DEV.035956

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6

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137

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41424687

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20150277

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2834461

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