about
ARID1a-DNA interactions are required for promoter occupancy by SWI/SNFStimulation of human and rat islet beta-cell proliferation with retention of function by the homeodomain transcription factor Nkx6.1FoxA2, Nkx2.2, and PDX-1 regulate islet beta-cell-specific mafA expression through conserved sequences located between base pairs -8118 and -7750 upstream from the transcription start siteThe Nkx6.1 homeodomain transcription factor suppresses glucagon expression and regulates glucose-stimulated insulin secretion in islet beta cells.Ataxia and hypogonadism caused by the loss of ubiquitin ligase activity of the U box protein CHIPMechanism of insulin gene regulation by the pancreatic transcription factor Pdx-1: application of pre-mRNA analysis and chromatin immunoprecipitation to assess formation of functional transcriptional complexes.Trefoil factor 3 stimulates human and rodent pancreatic islet beta-cell replication with retention of function.Isolation of a mycoplasma-specific binding peptide from an unbiased phage-displayed peptide library.Tobacco use induces anti-apoptotic, proliferative patterns of gene expression in circulating leukocytes of Caucasian males.Stable patterns of gene expression regulating carbohydrate metabolism determined by geographic ancestryMinireview: hey U(PS): metabolic and proteolytic homeostasis linked via AMPK and the ubiquitin proteasome system.Diggin' on u(biquitin): a novel method for the identification of physiological E3 ubiquitin ligase substrates.Regulation of AMPK by the ubiquitin proteasome systemCardio-metabolic effectsof HIV protease inhibitors (lopinavir/ritonavir)Coexistent ARID1A-PIK3CA mutations promote ovarian clear-cell tumorigenesis through pro-tumorigenic inflammatory cytokine signalling.Using community-based participatory research principles to develop more understandable recruitment and informed consent documents in genomic research.Muscle ring finger-3 protects against diabetic cardiomyopathy induced by a high fat diet.The ubiquitin ligase MuRF1 regulates PPARα activity in the heart by enhancing nuclear export via monoubiquitination.MuRF2 regulates PPARγ1 activity to protect against diabetic cardiomyopathy and enhance weight gain induced by a high fat diet.PHD2/3-dependent hydroxylation tunes cardiac response to β-adrenergic stress via phospholamban.Global gene expression profiling of a mouse model of ovarian clear cell carcinoma caused by ARID1A and PIK3CA mutations implicates a role for inflammatory cytokine signaling.Bmper inhibits endothelial expression of inflammatory adhesion molecules and protects against atherosclerosis.Fenofibrate unexpectedly induces cardiac hypertrophy in mice lacking MuRF1.Endothelial inflammatory transcriptional responses to an altered plasma exposome following inhalation of diesel emissionsEmerging evidence of coding mutations in the ubiquitin-proteasome system associated with cerebellar ataxiasA VGF-derived peptide attenuates development of type 2 diabetes via enhancement of islet β-cell survival and function.Pdx-1 activates islet α- and β-cell proliferation via a mechanism regulated by transient receptor potential cation channels 3 and 6 and extracellular signal-regulated kinases 1 and 2.Brothers and sisters: molecular insights into arterial-venous heterogeneity.Build it up-Tear it down: protein quality control in the cardiac sarcomere.Cardiac muscle ring finger-1 increases susceptibility to heart failure in vivo.Using Genetic Technologies To Reduce, Rather Than Widen, Health Disparities.New insights into bone morphogenetic protein signaling: focus on angiogenesis.Appetite for destruction: E3 ubiquitin-ligase protection in cardiac disease.Blood pressure-associated polymorphism controls ARHGAP42 expression via serum response factor DNA binding.Seek and destroy: the ubiquitin----proteasome system in cardiac disease.Minireview: Won't get fooled again: the nonmetabolic roles of peroxisome proliferator-activated receptors (PPARs) in the heart.An adenovirus vector for efficient RNA interference-mediated suppression of target genes in insulinoma cells and pancreatic islets of langerhans.Human amylin proteotoxicity impairs protein biosynthesis, and alters major cellular signaling pathways in the heart, brain and liver of humanized diabetic rat model in vivo.Cardiac energy dependence on glucose increases metabolites related to glutathione and activates metabolic genes controlled by mechanistic target of rapamycin.mTORtuous effect on the elastic heart
P50
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P50
description
hulumtues
@sq
researcher
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wetenschapper
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հետազոտող
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name
Jonathan Schisler
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Jonathan Schisler
@en
Jonathan Schisler
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Jonathan Schisler
@nl
Jonathan Schisler
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type
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Jonathan Schisler
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Jonathan Schisler
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Jonathan Schisler
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Jonathan Schisler
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Jonathan Schisler
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prefLabel
Jonathan Schisler
@ast
Jonathan Schisler
@en
Jonathan Schisler
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Jonathan Schisler
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Jonathan Schisler
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P106
P1153
35389970600
P21
P31
P496
0000-0001-7382-2783