Oncosis represents the main type of cell death in mouse models of cholestasis.
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Kruppel-like zinc finger protein Glis2 is essential for the maintenance of normal renal functionsPERK eIF2 alpha kinase is required to regulate the viability of the exocrine pancreas in mice.Macrophage-mediated phagocytosis of apoptotic cholangiocytes contributes to reversal of experimental biliary fibrosis.Lithocholic acid feeding results in direct hepato-toxicity independent of neutrophil function in miceThe Impact of Sterile Inflammation in Acute Liver Injury.Sustained phosphorylation of Bid is a marker for resistance to Fas-induced apoptosis during chronic liver diseases.Decreasing mitochondrial fission prevents cholestatic liver injuryLithocholic acid feeding induces segmental bile duct obstruction and destructive cholangitis in mice.Bile acid-induced necrosis in primary human hepatocytes and in patients with obstructive cholestasis.Effect of bile duct ligation on bile acid composition in mouse serum and liverNovel insight into mechanisms of cholestatic liver injury.Lysosomal instability and cathepsin B release during acetaminophen hepatotoxicity.Deficiency of iPLA₂β Primes Immune Cells for Proinflammation: Potential Involvement in Age-Related Mesenteric Lymph Node LymphomaCritical review of resveratrol in xenobiotic-induced hepatotoxicity.Drug insight: Mechanisms and sites of action of ursodeoxycholic acid in cholestasis.Critical Factors in the Assessment of Cholestatic Liver Injury In Vitro.Therapeutic targets for cholestatic liver injuryNIM811 (N-methyl-4-isoleucine cyclosporine), a mitochondrial permeability transition inhibitor, attenuates cholestatic liver injury but not fibrosis in mice.Bile-acid-induced cell injury and protectionThe BH3-only protein bid does not mediate death-receptor-induced liver injury in obstructive cholestasis.Plasma biomarkers of liver injury and inflammation demonstrate a lack of apoptosis during obstructive cholestasis in miceApoptosis and necrosis in the liverOsteopontin is an initial mediator of inflammation and liver injury during obstructive cholestasis after bile duct ligation in mice.Heparin-binding epidermal growth factor-like growth factor and hepatocyte growth factor inhibit cholestatic liver injury in mice through different mechanisms.Inflammatory pseudo-tumor of the liver: a rare pathological entityUrsodeoxycholic acid in cholestasis: linking action mechanisms to therapeutic applications.Disruption of the growth hormone--signal transducer and activator of transcription 5--insulinlike growth factor 1 axis severely aggravates liver fibrosis in a mouse model of cholestasis.Matrix metalloproteinase-10 expression is induced during hepatic injury and plays a fundamental role in liver tissue repair.Effects of bile acids on biliary epithelial cell proliferation and portal fibroblast activation using rat liver slices.Anti-apoptotic therapeutic approaches in liver diseases: do they really make sense?Absence of adipose triglyceride lipase protects from hepatic endoplasmic reticulum stress in mice.
P2860
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P2860
Oncosis represents the main type of cell death in mouse models of cholestasis.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Oncosis represents the main type of cell death in mouse models of cholestasis.
@en
Oncosis represents the main type of cell death in mouse models of cholestasis.
@nl
type
label
Oncosis represents the main type of cell death in mouse models of cholestasis.
@en
Oncosis represents the main type of cell death in mouse models of cholestasis.
@nl
prefLabel
Oncosis represents the main type of cell death in mouse models of cholestasis.
@en
Oncosis represents the main type of cell death in mouse models of cholestasis.
@nl
P2093
P50
P1476
Oncosis represents the main type of cell death in mouse models of cholestasis.
@en
P2093
Andrea Fuchsbichler
Hanns-Ulrich Marschall
Helmut Denk
Martin Wagner
Michael Trauner
P304
P356
10.1016/J.JHEP.2004.10.016
P577
2005-03-01T00:00:00Z