Multiwall carbon nanotubes mediate macrophage activation and promote pulmonary fibrosis through TGF-β/Smad signaling pathway.
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Effect of fiber length on carbon nanotube-induced fibrogenesisAdvances in mechanisms and signaling pathways of carbon nanotube toxicityIn vivo activation of a T helper 2-driven innate immune response in lung fibrosis induced by multi-walled carbon nanotubesNano-Therapeutics for the Lung: State-of-the-Art and Future PerspectivesRight or left: the role of nanoparticles in pulmonary diseasesTIMP1 promotes multi-walled carbon nanotube-induced lung fibrosis by stimulating fibroblast activation and proliferationMyofibroblasts and lung fibrosis induced by carbon nanotube exposureMechanisms of lung fibrosis induced by carbon nanotubes: towards an Adverse Outcome Pathway (AOP)Nanometer-long Ge-imogolite nanotubes cause sustained lung inflammation and fibrosis in ratsInflammasome activation in airway epithelial cells after multi-walled carbon nanotube exposure mediates a profibrotic response in lung fibroblastsNano-risk Science: application of toxicogenomics in an adverse outcome pathway framework for risk assessment of multi-walled carbon nanotubesTowards predicting the lung fibrogenic activity of nanomaterials: experimental validation of an in vitro fibroblast proliferation assayHelical carbon nanotubes enhance the early immune response and inhibit macrophage-mediated phagocytosis of Pseudomonas aeruginosaIL-33 modulates chronic airway resistance changes induced by multi-walled carbon nanotubesMulti-walled carbon nanotubes directly induce epithelial-mesenchymal transition in human bronchial epithelial cells via the TGF-β-mediated Akt/GSK-3β/SNAIL-1 signalling pathwayMultiwalled Carbon Nanotube Functionalization with High Molecular Weight Hyaluronan Significantly Reduces Pulmonary InjuryLong-term intravenous administration of carboxylated single-walled carbon nanotubes induces persistent accumulation in the lungs and pulmonary fibrosis via the nuclear factor-kappa B pathwaySTAT1-dependent and -independent pulmonary allergic and fibrogenic responses in mice after exposure to tangled versus rod-like multi-walled carbon nanotubesRoles of TGF-β/Smad signaling pathway in pathogenesis and development of gluteal muscle contracture.Cross regulation between hypoxia-inducible transcription factor-1α (HIF-1α) and transforming growth factor (TGF)-ß1 mediates nickel oxide nanoparticles (NiONPs)-induced pulmonary fibrosisToxicity assessment and bioaccumulation in zebrafish embryos exposed to carbon nanotubes suspended in Pluronic® F-108.Ginsenoside Rg1 Ameliorates Cigarette Smoke-Induced Airway Fibrosis by Suppressing the TGF-β1/Smad Pathway In Vivo and In Vitro.Impact and effectiveness of risk mitigation strategies on the insurability of nanomaterial production: evidences from industrial case studies.Review of toxicity studies of carbon nanotubesSimple in vitro models can predict pulmonary toxicity of silver nanoparticles.Predicting pulmonary fibrosis in humans after exposure to multi-walled carbon nanotubes (MWCNTs).Activation of Wnt/β-catenin signalling is required for TGF-β/Smad2/3 signalling during myofibroblast proliferation.Multiwall carbon nanotubes directly promote fibroblast-myofibroblast and epithelial-mesenchymal transitions through the activation of the TGF-β/Smad signaling pathway.High-dose dexamethasone induced LPS-stimulated rat alveolar macrophages apoptosis.Mechanisms of carbon nanotube-induced pulmonary fibrosis: a physicochemical characteristic perspective.Mediation of the single-walled carbon nanotubes induced pulmonary fibrogenic response by osteopontin and TGF-β1.Structure Activity Relationships of Engineered Nanomaterials in inducing NLRP3 Inflammasome Activation and Chronic Lung Fibrosis.Dioscin Exerts Protective Effects Against Crystalline Silica-induced Pulmonary Fibrosis in Mice.Bacterial endotoxin (lipopolysaccharide) binds to the surface of gold nanoparticles, interferes with biocorona formation and induces human monocyte inflammatory activation.Diameter-dependent release of a cisplatin pro-drug from small and large functionalized carbon nanotubes.Nickel oxide nanoparticles induced pulmonary fibrosis via TGF- β1 activation in rats.Early-life exposure to three size-fractionated ultrafine and fine atmospheric particulates in Beijing exacerbates asthma development in mature mice.Towards predicting the lung fibrogenic activity of MWCNT: Key role of endocytosis, kinase receptors and ERK 1/2 signaling.Type 2 Immune Mechanisms in Carbon Nanotube-Induced Lung Fibrosis.The Toxic Truth About Carbon Nanotubes in Water Purification: a Perspective View.
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P2860
Multiwall carbon nanotubes mediate macrophage activation and promote pulmonary fibrosis through TGF-β/Smad signaling pathway.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh-hant
name
Multiwall carbon nanotubes med ...... TGF-β/Smad signaling pathway.
@en
Multiwall carbon nanotubes med ...... TGF-β/Smad signaling pathway.
@nl
type
label
Multiwall carbon nanotubes med ...... TGF-β/Smad signaling pathway.
@en
Multiwall carbon nanotubes med ...... TGF-β/Smad signaling pathway.
@nl
prefLabel
Multiwall carbon nanotubes med ...... TGF-β/Smad signaling pathway.
@en
Multiwall carbon nanotubes med ...... TGF-β/Smad signaling pathway.
@nl
P2093
P2860
P50
P356
P1433
P1476
Multiwall carbon nanotubes med ...... TGF-β/Smad signaling pathway.
@en
P2093
Chunying Chen
Lili Zhang
Yuliang Zhao
Zhiyun Chen
P2860
P304
P356
10.1002/SMLL.201300607
P577
2013-05-06T00:00:00Z