Structure Activity Relationships of Engineered Nanomaterials in inducing NLRP3 Inflammasome Activation and Chronic Lung Fibrosis.

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Review of techniques and studies characterizing the release of carbon nanotubes from nanocomposites: Implications for exposure and human health risk assessment.

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Structure Activity Relationships of Engineered Nanomaterials in inducing NLRP3 Inflammasome Activation and Chronic Lung Fibrosis.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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Structure Activity Relationshi ...... ion and Chronic Lung Fibrosis.

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Structure Activity Relationshi ...... ion and Chronic Lung Fibrosis.

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Structure Activity Relationshi ...... ion and Chronic Lung Fibrosis.

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Structure Activity Relationshi ...... ion and Chronic Lung Fibrosis.

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Structure Activity Relationshi ...... ion and Chronic Lung Fibrosis.

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Structure Activity Relationshi ...... ion and Chronic Lung Fibrosis.

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Structure Activity Relationshi ...... ion and Chronic Lung Fibrosis.

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P2093

Bingbing Sun

http://www.w3.org/2001/XMLSchema#string

Sijin Liu

http://www.w3.org/2001/XMLSchema#string

Xiang Wang

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P2860

Inhalation vs. aspiration of single-walled carbon nanotubes in C57BL/6 mice: inflammation, fibrosis, oxidative stress, and mutagenesis

Multi-walled carbon nanotubes: sampling criteria and aerosol characterization

Interlaboratory evaluation of rodent pulmonary responses to engineered nanomaterials: the NIEHS Nano Go Consortium

Direct fibrogenic effects of dispersed single-walled carbon nanotubes on human lung fibroblasts

Decreased dissolution of ZnO by iron doping yields nanoparticles with reduced toxicity in the rodent lung and zebrafish embryos

Quantitative techniques for assessing and controlling the dispersion and biological effects of multiwalled carbon nanotubes in mammalian tissue culture cells

Dispersal state of multiwalled carbon nanotubes elicits profibrogenic cellular responses that correlate with fibrogenesis biomarkers and fibrosis in the murine lung

The inflammasomes

Nanotoxicology: an emerging discipline evolving from studies of ultrafine particles

Nanoparticles activate the NLR pyrin domain containing 3 (Nlrp3) inflammasome and cause pulmonary inflammation through release of IL-1α and IL-1β

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99-108

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scholarly article

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10.1016/J.IMPACT.2016.08.002

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P478

http://www.w3.org/2001/XMLSchema#string

P50

Tian Xia

P577

2016-08-20T00:00:00Z

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P698

28480337

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pulmonary fibrosis

inflammasome

nanomaterial

P932

5415341

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Structure Activity Relationships of Engineered Nanomaterials in inducing NLRP3 Inflammasome Activation and Chronic Lung Fibrosis.

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Structure Activity Relationships of Engineered Nanomaterials in inducing NLRP3 Inflammasome Activation and Chronic Lung Fibrosis.

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P2860

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P356

P478

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P577

P698

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P2093

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P304

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P577

P698

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