Transition in the mechanism of flow-mediated dilation with aging and development of coronary artery disease.

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Evaluation of Vascular Control Mechanisms Utilizing Video Microscopy of Isolated Resistance Arteries of Rats.Contribution of mitochondria-derived free radicals to endothelial dysfunction in human skeletal muscle feed arteries: another hazard of the ageing process.

P2860

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Transition in the mechanism of flow-mediated dilation with aging and development of coronary artery disease.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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Transition in the mechanism of ...... nt of coronary artery disease.

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Transition in the mechanism of ...... nt of coronary artery disease.

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Transition in the mechanism of ...... nt of coronary artery disease.

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Transition in the mechanism of ...... nt of coronary artery disease.

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Transition in the mechanism of ...... nt of coronary artery disease.

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Transition in the mechanism of ...... nt of coronary artery disease.

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Transition in the mechanism of ...... ent of coronary artery disease

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April L Wittenburg

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Bradley Miller

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David D Gutterman

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Michael Mitchell

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Natalya Zinkevich

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Ralph Galdieri

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Yanping Liu

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P2860

Differential regulation of TRPV1 channels by H2O2: implications for diabetic microvascular dysfunction.

Ceramide changes the mediator of flow-induced vasodilation from nitric oxide to hydrogen peroxide in the human microcirculation.

Altered cutaneous microvascular responses to reactive hyperaemia in coronary artery disease: a comparative study with conduit vessel responses.

The vascular renin-angiotensin system contributes to blunted vasodilation induced by transient high pressure in human adipose microvessels

Endothelium dependency of contractile activity differs in infant and adult vertebral arteries

Endothelium-mediated coronary blood flow modulation in humans. Effects of age, atherosclerosis, hypercholesterolemia, and hypertension.

Mechanosensitive PPAP2B Regulates Endothelial Responses to Atherorelevant Hemodynamic Forces.

An acute rise in intraluminal pressure shifts the mediator of flow-mediated dilation from nitric oxide to hydrogen peroxide in human arterioles.

H2O2 is the transferrable factor mediating flow-induced dilation in human coronary arterioles

Reactive oxygen species in the vasculature: molecular and cellular mechanisms.

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s00395-016-0594-x

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scholarly article

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10.1007/S00395-016-0594-X

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Andrey Sorokin

Andreas M Beyer

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2016-12-19T00:00:00Z

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Transition in the mechanism of flow-mediated dilation with aging and development of coronary artery disease.

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P2860

P2860

Transition in the mechanism of flow-mediated dilation with aging and development of coronary artery disease.

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