Nrf2/antioxidant pathway mediates β cell self-repair after damage by high-fat diet-induced oxidative stress.

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β-Cell Autophagy in Diabetes Pathogenesis.Intermittent high glucose-induced oxidative stress modulates retinal pigmented epithelial cell autophagy and promotes cell survival via increased HMGB1

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Nrf2/antioxidant pathway mediates β cell self-repair after damage by high-fat diet-induced oxidative stress.

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Nrf2/antioxidant pathway media ...... diet-induced oxidative stress.

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Nrf2/antioxidant pathway media ...... diet-induced oxidative stress.

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Nrf2/antioxidant pathway media ...... diet-induced oxidative stress.

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Nrf2/antioxidant pathway media ...... diet-induced oxidative stress.

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Nrf2/antioxidant pathway media ...... diet-induced oxidative stress.

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Nrf2/antioxidant pathway media ...... diet-induced oxidative stress.

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Nrf2/antioxidant pathway media ...... diet-induced oxidative stress.

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Elizabeth Oseid

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Fumihiko Urano

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Jana Mahadevan

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Lindsey Bogachus

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Michele Black

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R Paul Robertson

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Stephanie Hahn

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Tsehay Abebe

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Vincenzo Cirulli

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P2860

Epigenetic regulation of Keap1-Nrf2 signaling

Mechanisms of activation of the transcription factor Nrf2 by redox stressors, nutrient cues, and energy status and the pathways through which it attenuates degenerative disease

Beta-cell deficit and increased beta-cell apoptosis in humans with type 2 diabetes

Autophagy defends pancreatic β cells from human islet amyloid polypeptide-induced toxicity

Minireview: Thioredoxin-interacting protein: regulation and function in the pancreatic β-cell.

Reversible changes in pancreatic islet structure and function produced by elevated blood glucose.

Chronic exposure of HIT cells to high glucose concentrations paradoxically decreases insulin gene transcription and alters binding of insulin gene regulatory protein.

A role for glutathione peroxidase in protecting pancreatic beta cells against oxidative stress in a model of glucose toxicity.

Oxidative stress-mediated, post-translational loss of MafA protein as a contributing mechanism to loss of insulin gene expression in glucotoxic beta cells.

Glucotoxic and diabetic conditions induce caspase 6-mediated degradation of nuclear lamin A in human islets, rodent islets and INS-1 832/13 cells

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scholarly article

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10.1172/JCI.INSIGHT.92854

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Nrf2/antioxidant pathway mediates β cell self-repair after damage by high-fat diet-induced oxidative stress.

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P2860

P2860

Nrf2/antioxidant pathway mediates β cell self-repair after damage by high-fat diet-induced oxidative stress.

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