Ethanol Consumption in Mice Lacking CD14, TLR2, TLR4, or MyD88.
about
Genetic and Pharmacologic Manipulation of TLR4 Has Minimal Impact on Ethanol Consumption in Rodents.CNS cell-type localization and LPS response of TLR signaling pathwaysInnately activated TLR4 signal in the nucleus accumbens is sustained by CRF amplification loop and regulates impulsivity.Sedative and Motor Incoordination Effects of Ethanol in Mice Lacking CD14, TLR2, TLR4, or MyD88.Chronic ethanol consumption: role of TLR3/TRIF-dependent signaling.Role of TLR4 in the Modulation of Central Amygdala GABA Transmission by CRF Following Restraint Stress.Apremilast Alters Behavioral Responses to Ethanol in Mice: II. Increased Sedation, Intoxication, and Reduced Acute Functional Tolerance.The GABAA Receptor α2 Subunit Activates a Neuronal TLR4 Signal in the Ventral Tegmental Area that Regulates Alcohol and Nicotine Abuse.Damage-associated molecular pattern recognition is required for induction of retinal neuroprotective pathways in a sex-dependent manner.Deep sequencing and miRNA profiles in alcohol-induced neuroinflammation and the TLR4 response in mice cerebral cortex
P2860
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P2860
Ethanol Consumption in Mice Lacking CD14, TLR2, TLR4, or MyD88.
description
2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年學術文章
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name
Ethanol Consumption in Mice Lacking CD14, TLR2, TLR4, or MyD88.
@en
Ethanol Consumption in Mice Lacking CD14, TLR2, TLR4, or MyD88.
@nl
type
label
Ethanol Consumption in Mice Lacking CD14, TLR2, TLR4, or MyD88.
@en
Ethanol Consumption in Mice Lacking CD14, TLR2, TLR4, or MyD88.
@nl
prefLabel
Ethanol Consumption in Mice Lacking CD14, TLR2, TLR4, or MyD88.
@en
Ethanol Consumption in Mice Lacking CD14, TLR2, TLR4, or MyD88.
@nl
P2093
P2860
P356
P1476
Ethanol Consumption in Mice Lacking CD14, TLR2, TLR4, or MyD88.
@en
P2093
Adriana Da Costa
Jody Mayfield
Julia Chernis
Mendy Black
R Adron Harris
Yuri A Blednov
P2860
P304
P356
10.1111/ACER.13316
P577
2017-02-01T00:00:00Z