HDAC4: a key factor underlying brain developmental alterations in CDKL5 disorder.
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Searching for biomarkers of CDKL5 disorder: early-onset visual impairment in CDKL5 mutant mice.Loss of CDKL5 in Glutamatergic Neurons Disrupts Hippocampal Microcircuitry and Leads to Memory Impairment in Mice.The neurosteroid pregnenolone reverts microtubule derangement induced by the loss of a functional CDKL5-IQGAP1 complex.Comprehensive behavioral analysis of the Cdkl5 knockout mice revealed significant enhancement in anxiety- and fear-related behaviors and impairment in both acquisition and long-term retention of spatial reference memory.Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder.
P2860
HDAC4: a key factor underlying brain developmental alterations in CDKL5 disorder.
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2016 nî lūn-bûn
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2016年の論文
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HDAC4: a key factor underlying brain developmental alterations in CDKL5 disorder.
@en
HDAC4: a key factor underlying brain developmental alterations in CDKL5 disorder.
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HDAC4: a key factor underlying brain developmental alterations in CDKL5 disorder.
@en
HDAC4: a key factor underlying brain developmental alterations in CDKL5 disorder.
@nl
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HDAC4: a key factor underlying brain developmental alterations in CDKL5 disorder.
@en
HDAC4: a key factor underlying brain developmental alterations in CDKL5 disorder.
@nl
P2093
P2860
P50
P356
P1476
HDAC4: a key factor underlying brain developmental alterations in CDKL5 disorder.
@en
P2093
Claudia Fuchs
Giovanni Perini
Renata Bartesaghi
Roberto Rimondini
Rocchina Viggiano
Stefania Trazzi
Thomas Kurz
P2860
P304
P356
10.1093/HMG/DDW231
P577
2016-07-27T00:00:00Z