TL1A produced by lamina propria macrophages induces Th1 and Th17 immune responses in cooperation with IL-23 in patients with Crohn's disease.
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The TNF-family cytokine TL1A: from lymphocyte costimulator to disease co-conspiratorInterleukin-18 genetic polymorphisms contribute differentially to the susceptibility to Crohn's diseaseIntestinal Antigen-Presenting Cells: Key Regulators of Immune Homeostasis and InflammationDecoy receptor 3: an endogenous immunomodulator in cancer growth and inflammatory reactions.Tumour necrosis factor superfamily members in the pathogenesis of inflammatory bowel diseaseCX₃CR1⁺ mononuclear phagocytes support colitis-associated innate lymphoid cell production of IL-22.Secukinumab, a human anti-IL-17A monoclonal antibody, for moderate to severe Crohn's disease: unexpected results of a randomised, double-blind placebo-controlled trial.Use of animal models in elucidating disease pathogenesis in IBD.The TNF-family cytokine TL1A inhibits proliferation of human activated B cellsStrong viremia control in vaccinated macaques does not prevent gradual Th17 cell loss from central memory.A major population of mucosal memory CD4+ T cells, coexpressing IL-18Rα and DR3, display innate lymphocyte functionalityCD161 DEFINES EFFECTOR T CELLS THAT EXPRESS LIGHT AND RESPOND TO TL1A-DR3 SIGNALING.TL1A/DR3 axis involvement in the inflammatory cytokine network during pulmonary sarcoidosis.IL12p40 regulates functional development of human CD4+ T cells: enlightenment by the elevated expressions of IL12p40 in patients with inflammatory bowel diseasesChemokine and cytokine levels in inflammatory bowel disease patients.Role of resveratrol-induced CD11b(+) Gr-1(+) myeloid derived suppressor cells (MDSCs) in the reduction of CXCR3(+) T cells and amelioration of chronic colitis in IL-10(-/-) mice.SUSTAINED TL1A (TNFSF15) EXPRESSION ON BOTH LYMPHOID AND MYELOID CELLS LEADS TO MILD SPONTANEOUS INTESTINAL INFLAMMATION AND FIBROSIS.High intestinal and systemic levels of interleukin-23/T-helper 17 pathway in Chinese patients with inflammatory bowel disease.The role of TL1A and DR3 in autoimmune and inflammatory diseases.Multiple activating and repressive cis-promoter regions regulate TNFSF15 expression in human primary mononuclear cells.Proinflammatory cytokines underlying the inflammation of Crohn's diseaseTh17-cytokine blockers as a new approach for treating inflammatory bowel disease.Genome-wide association studies and Crohn's disease.Changes of the cytokine profile in inflammatory bowel diseases.The unusual suspects--innate lymphoid cells as novel therapeutic targets in IBD.Tumor Necrosis Factor-like Cytokine TL1A and Its Receptors DR3 and DcR3: Important New Factors in Mucosal Homeostasis and Inflammation.Genetic Markers Associated with Clinical Outcomes in Patients with Inflammatory Bowel Disease.Morinda citrifolia (Noni) Fruit Juice Reduces Inflammatory Cytokines Expression and Contributes to the Maintenance of Intestinal Mucosal Integrity in DSS Experimental Colitis.Multifaceted death receptor 3 signaling-promoting survival and triggering death.TL1A as a Potential Local Inducer of IL17A Expression in Colon Mucosa of Inflammatory Bowel Disease Patients.The death receptor 3/TL1A pathway is essential for efficient development of antiviral CD4⁺ and CD8⁺ T-cell immunity.Elevated risk of opportunistic viral infection in patients with Crohn's disease during biological therapies: a meta analysis of randomized controlled trials.A Novel Role of Spred2 in the Colonic Epithelial Cell Homeostasis and Inflammation.Myosin light chain kinase inhibitor inhibits dextran sulfate sodium-induced colitis in mice.Mucosal CXCR4+ IgG plasma cells contribute to the pathogenesis of human ulcerative colitis through FcγR-mediated CD14 macrophage activation.Identifying and testing candidate genetic polymorphisms in the irritable bowel syndrome (IBS): association with TNFSF15 and TNFα.Expression of death receptor 3 on peripheral blood mononuclear cells differes in adult IBD patients and children with newly diagnosed IBD.Reduced numbers of mucosal DR(int) macrophages and increased numbers of CD103(+) dendritic cells during anti-TNF-α treatment in patients with Crohn's disease.The Role of T-Cell Subsets in Chronic Inflammation in Celiac Disease and Inflammatory Bowel Disease Patients: More Common Mechanisms or More Differences?Smoking behaviour modifiesIL23r-associated disease risk in patients with Crohn's disease
P2860
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P2860
TL1A produced by lamina propria macrophages induces Th1 and Th17 immune responses in cooperation with IL-23 in patients with Crohn's disease.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh-hant
name
TL1A produced by lamina propri ...... patients with Crohn's disease.
@en
TL1A produced by lamina propri ...... patients with Crohn's disease.
@nl
type
label
TL1A produced by lamina propri ...... patients with Crohn's disease.
@en
TL1A produced by lamina propri ...... patients with Crohn's disease.
@nl
prefLabel
TL1A produced by lamina propri ...... patients with Crohn's disease.
@en
TL1A produced by lamina propri ...... patients with Crohn's disease.
@nl
P2093
P2860
P356
P1476
TL1A produced by lamina propri ...... patients with Crohn's disease.
@en
P2093
Akira Sugita
Haruki Honda
Hiroshi Chinen
Kazutaka Koganei
Mina T Kitazume
Susumu Okamoto
Tadakazu Hisamatsu
Takanori Kanai
Taku Kobayashi
Tetsuro Takayama
P2860
P304
P356
10.1002/IBD.21124
P577
2010-04-01T00:00:00Z