about
The K-Cl cotransporter KCC3 is mutant in a severe peripheral neuropathy associated with agenesis of the corpus callosumVitamin C deficiency increases basal exploratory activity but decreases scopolamine-induced activity in APP/PSEN1 transgenic miceGalanin-acetylcholine interactions in rodent memory tasks and Alzheimer's disease.Intracranial V. cholerae sialidase protects against excitotoxic neurodegenerationDiscrimination of methadone and cocaine by pigeons without explicit discrimination trainingGanglioside metabolism in a transgenic mouse model of Alzheimer's disease: expression of Chol-1α antigens in the brain.Abnormal vibrissa-related behavior and loss of barrel field inhibitory neurons in 5xFAD transgenics.4-Caffeoyl-1,5-quinide in roasted coffee inhibits [3H]naloxone binding and reverses anti-nociceptive effects of morphine in mice.A single intramuscular injection of rAAV-mediated mutant erythropoietin protects against MPTP-induced parkinsonism.Role of ganglioside metabolism in the pathogenesis of Alzheimer's disease--a reviewElevated oxidative stress and sensorimotor deficits but normal cognition in mice that cannot synthesize ascorbic acidRetinoic acid attenuates beta-amyloid deposition and rescues memory deficits in an Alzheimer's disease transgenic mouse model.Reversal of beta-amyloid-induced retention deficit after exposure to training and state cues.Effects of an exogenous beta-amyloid peptide on retention for spatial learning.Coadministration of galanin antagonist M40 with a muscarinic M1 agonist improves delayed nonmatching to position choice accuracy in rats with cholinergic lesions.Hyperactivity, impaired learning on a vigilance task, and a differential response to methylphenidate in the TRbetaPV knock-in mouse.Norepinephrine transporter-deficient mice respond to anxiety producing and fearful environments with bradycardia and hypotension.Expression of the mutant thyroid hormone receptor PV in the pituitary of transgenic mice leads to weight reduction.Biochemical and morphometric analyses show that myelination in the insulin-like growth factor 1 null brain is proportionate to its neuronal composition.Targeted deletion of GD3 synthase protects against MPTP-induced neurodegeneration.Behavioral effects of ivermectin in mice.Impaired spatial learning in the APPSwe + PSEN1DeltaE9 bigenic mouse model of Alzheimer's disease.Transgenic mice expressing a human mutant beta1 thyroid receptor are hyperactive, impulsive, and inattentive.Intrahippocampal injections of exogenous beta-amyloid induce postdelay errors in an eight-arm radial maze.Elimination of GD3 synthase improves memory and reduces amyloid-beta plaque load in transgenic mice.Impaired spatial memory in APP-overexpressing mice on a homocysteinemia-inducing diet.Abnormal GABAA receptor-mediated currents in dorsal root ganglion neurons isolated from Na-K-2Cl cotransporter null mice.
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description
American neurologist
@en
neurólogo estadounidense
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onderzoeker
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name
Michael P McDonald
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Michael P McDonald
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Michael P McDonald
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Michael P. McDonald
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type
label
Michael P McDonald
@es
Michael P McDonald
@nl
Michael P McDonald
@sl
Michael P. McDonald
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prefLabel
Michael P McDonald
@es
Michael P McDonald
@nl
Michael P McDonald
@sl
Michael P. McDonald
@en
P1960
i8qTv-AAAAAJ
P21
P31
P496
0000-0002-9642-964X