about
Convergent transcription at intragenic super-enhancers targets AID-initiated genomic instabilityDevelopmental propagation of V(D)J recombination-associated DNA breaks and translocations in mature B cells via dicentric chromosomes.Genome-wide detection of DNA double-stranded breaks induced by engineered nucleases.Detecting DNA double-stranded breaks in mammalian genomes by linear amplification-mediated high-throughput genome-wide translocation sequencingOrientation-specific joining of AID-initiated DNA breaks promotes antibody class switching.Long Neural Genes Harbor Recurrent DNA Break Clusters in Neural Stem/Progenitor Cells.Transcription-associated processes cause DNA double-strand breaks and translocations in neural stem/progenitor cells.Microbial colonization influences early B-lineage development in the gut lamina propria.IgH class switching exploits a general property of two DNA breaks to be joined in cis over long chromosomal distances.Sequence-Intrinsic Mechanisms that Target AID Mutational Outcomes on Antibody GenesGenomic Copy Number Dictates a Gene-Independent Cell Response to CRISPR/Cas9 Targeting.Chromosomal Loop Domains Direct the Recombination of Antigen Receptor Genes.Computational correction of copy number effect improves specificity of CRISPR-Cas9 essentiality screens in cancer cells.Targetable vulnerabilities in T- and NK-cell lymphomas identified through preclinical models.
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description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Robin M Meyers
@ast
Robin M Meyers
@en
Robin M Meyers
@es
Robin M Meyers
@nl
type
label
Robin M Meyers
@ast
Robin M Meyers
@en
Robin M Meyers
@es
Robin M Meyers
@nl
prefLabel
Robin M Meyers
@ast
Robin M Meyers
@en
Robin M Meyers
@es
Robin M Meyers
@nl
P106
P31
P496
0000-0002-1802-7231