about
Importance of prophages to evolution and virulence of bacterial pathogensNovel riboswitch ligand analogs as selective inhibitors of guanine-related metabolic pathwaysFaecal pharmacokinetics of orally administered vancomycin in patients with suspected Clostridium difficile infection.c-di-GMP turn-over in Clostridium difficile is controlled by a plethora of diguanylate cyclases and phosphodiesterases.Cyclic di-GMP riboswitch-regulated type IV pili contribute to aggregation of Clostridium difficile.Function of the CRISPR-Cas System of the Human Pathogen Clostridium difficile.Factors Associated With Complications of Clostridium difficile Infection in a Multicenter Prospective CohortPrevention of Clostridium difficile spore formation by sub-inhibitory concentrations of tigecycline and piperacillin/tazobactam.PhageTerm: a tool for fast and accurate determination of phage termini and packaging mechanism using next-generation sequencing data.Characterization of temperate phages infecting Clostridium difficile isolates of human and animal origins.A sexually dichotomous, autistic-like phenotype is induced by Group B Streptococcus maternofetal immune activation.Antiapoptotic proteins Bcl-2 and Bcl-XL inhibit Clostridium difficile toxin A-induced cell death in human epithelial cells.Ileal antimicrobial peptide expression is dysregulated in old age.Activation of the IL-1β/CXCL1/MMP-10 axis in chorioamnionitis induced by inactivated Group B Streptococcus.Characterization of Functional Prophages in Clostridium difficile.The Clostridium difficile cell wall protein CwpV confers phase-variable phage resistance.Evidence of in vivo prophage induction during Clostridium difficile infection.Global transcriptional response of Clostridium difficile carrying the CD38 prophage.White matter injury and autistic-like behavior predominantly affecting male rat offspring exposed to group B streptococcal maternal inflammation.Prophage-stimulated toxin production in Clostridium difficile NAP1/027 lysogens.Erratum for Boudry et al., function of the CRISPR-Cas system of the human pathogen Clostridium difficile.AbiV, a novel antiphage abortive infection mechanism on the chromosome of Lactococcus lactis subsp. cremoris MG1363.Identification of common polymorphisms in the promoter of the UGT1A9 gene: evidence that UGT1A9 protein and activity levels are strongly genetically controlled in the liver.The Contribution of Bacteriophages to the Biology and Virulence of Pathogenic Clostridia.High prevalence and genetic diversity of large phiCD211/phiCDIF1296T-like prophages in Clostridioides difficile.Purine analogs targeting the guanine riboswitch as potential antibiotics against Clostridioides difficile.Bacteriophages Contribute to Shaping SpeciesTHE NOVEL UGT1A9 INTRONIC I399 POLYMORPHISM APPEARS AS A PREDICTOR OF 7-ETHYL-10-HYDROXYCAMPTOTHECIN GLUCURONIDATION LEVELS IN THE LIVERAcid sensitivity of neomycin-resistant mutants of Oenococcus oeni: a relationship between reduction of ATPase activity and lack of malolactic activityInduction of Oenococcus oeni H+-ATPase activity and mRNA transcription under acidic conditions[Discovery of keyicin, a new antibiotic: it takes two to tango]Multilocus Variable-Number Tandem-Repeat Analysis of Clostridioides difficile Clusters in Ribotype 027 Isolates and Lack of Association with Clinical Outcomes
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description
researcher
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wetenschapper
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հետազոտող
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name
Louis-Charles Fortier
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Louis-Charles Fortier
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Louis-Charles Fortier
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Louis-Charles Fortier
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type
label
Louis-Charles Fortier
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Louis-Charles Fortier
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Louis-Charles Fortier
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Louis-Charles Fortier
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prefLabel
Louis-Charles Fortier
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Louis-Charles Fortier
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Louis-Charles Fortier
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Louis-Charles Fortier
@nl
P106
P31
P496
0000-0003-1950-763X