about
Bardet-Biedl syndrome 3 (Bbs3) knockout mouse model reveals common BBS-associated phenotypes and Bbs3 unique phenotypesRequirement of Bardet-Biedl syndrome proteins for leptin receptor signalingBMP8B increases brown adipose tissue thermogenesis through both central and peripheral actionsThe brain and brown fatEctopic expression of human BBS4 can rescue Bardet-Biedl syndrome phenotypes in Bbs4 null miceGlucose depletion in the airway surface liquid is essential for sterility of the airwaysA mitochondrial-targeted coenzyme q analog prevents weight gain and ameliorates hepatic dysfunction in high-fat-fed miceThe ion channel ASIC2 is required for baroreceptor and autonomic control of the circulation.Mkks-null mice have a phenotype resembling Bardet-Biedl syndromeLeptin resistance contributes to obesity and hypertension in mouse models of Bardet-Biedl syndromeSIRT1 deacetylase in POMC neurons is required for homeostatic defenses against diet-induced obesityA knockin mouse model of the Bardet-Biedl syndrome 1 M390R mutation has cilia defects, ventriculomegaly, retinopathy, and obesityCiliopathy is differentially distributed in the brain of a Bardet-Biedl syndrome mouse modelMetabolic control by S6 kinases depends on dietary lipidsContrasting vascular effects caused by loss of Bardet-Biedl syndrome genes.The cellular and molecular bases of leptin and ghrelin resistance in obesity.Selective Deletion of the Brain-Specific Isoform of Renin Causes Neurogenic Hypertension.Sympathetic tone in the young: the mother weighs in.Traveling from the hypothalamus to the adipose tissue: The thermogenic pathwayEstradiol regulates brown adipose tissue thermogenesis via hypothalamic AMPKHypothalamic AMPK-ER Stress-JNK1 Axis Mediates the Central Actions of Thyroid Hormones on Energy Balance.Brain mineralocorticoid receptors and centrally regulated functions.Interference with peroxisome proliferator-activated receptor-γ in vascular smooth muscle causes baroreflex impairment and autonomic dysfunction.Differential effects of insulin on sympathetic nerve activity in agouti obese mice.Hypothalamic AMPK and fatty acid metabolism mediate thyroid regulation of energy balance.Three months of high-fructose feeding fails to induce excessive weight gain or leptin resistance in miceObesity-associated hypertension: recent progress in deciphering the pathogenesisFGF21 acts centrally to induce sympathetic nerve activity, energy expenditure, and weight loss.The brain Renin-angiotensin system controls divergent efferent mechanisms to regulate fluid and energy balance.Obesity-associated hypertension: new insights into mechanisms.Cardiovascular and sympathetic effects of leptin.Involvement of hypothalamic AMP-activated protein kinase in leptin-induced sympathetic nerve activation.Inactivation of Bardet-Biedl syndrome genes causes kidney defects.Neuronal receptor activity-modifying protein 1 promotes energy expenditure in mice.Selective leptin resistance: a new concept in leptin physiology with cardiovascular implications.Cardiovascular and sympathetic effects of disrupting tyrosine 985 of the leptin receptor.Differential Control of the Sympathetic Nervous System by Leptin: Implications for Obesity.Leptin receptor signaling in the hypothalamus regulates hepatic autonomic nerve activity via phosphatidylinositol 3-kinase and AMP-activated protein kinaseMolecular basis of the obesity associated with Bardet-Biedl syndrome.The role of hypothalamic mTORC1 signaling in insulin regulation of food intake, body weight, and sympathetic nerve activity in male mice.
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Kamal Rahmouni
@ast
Kamal Rahmouni
@en
Kamal Rahmouni
@es
Kamal Rahmouni
@nl
type
label
Kamal Rahmouni
@ast
Kamal Rahmouni
@en
Kamal Rahmouni
@es
Kamal Rahmouni
@nl
prefLabel
Kamal Rahmouni
@ast
Kamal Rahmouni
@en
Kamal Rahmouni
@es
Kamal Rahmouni
@nl
P108
P106
P1153
7003780122
P31
P496
0000-0001-5136-6748