about
α-Synuclein protects against manganese neurotoxic insult during the early stages of exposure in a dopaminergic cell model of Parkinson's diseaseFyn Kinase Regulates Microglial Neuroinflammatory Responses in Cell Culture and Animal Models of Parkinson's DiseaseCopper-induced structural conversion templates prion protein oligomerization and neurotoxicity.Effect of divalent metals on the neuronal proteasomal system, prion protein ubiquitination and aggregation.Molecular cloning, epigenetic regulation, and functional characterization of Prkd1 gene promoter in dopaminergic cell culture models of Parkinson's disease.The peptidyl-prolyl isomerase Pin1 up-regulation and proapoptotic function in dopaminergic neurons: relevance to the pathogenesis of Parkinson disease.Role of proteolytic activation of protein kinase Cδ in the pathogenesis of prion disease.Protein kinase Cδ upregulation in microglia drives neuroinflammatory responses and dopaminergic neurodegeneration in experimental models of Parkinson's disease.Prokineticin-2 upregulation during neuronal injury mediates a compensatory protective response against dopaminergic neuronal degeneration.Manganese exposure induces neuroinflammation by impairing mitochondrial dynamics in astrocytes.Neuronal protection against oxidative insult by polyanhydride nanoparticle-based mitochondria-targeted antioxidant therapy.Role of neurotoxicants and traumatic brain injury in α-synuclein protein misfolding and aggregation.Exosomes in Toxicology: Relevance to Chemical Exposure and Pathogenesis of Environmentally Linked Diseases.Mitoapocynin Treatment Protects Against Neuroinflammation and Dopaminergic Neurodegeneration in a Preclinical Animal Model of Parkinson's Disease.Mitochondrial impairment in microglia amplifies NLRP3 inflammasome proinflammatory signaling in cell culture and animal models of Parkinson's disease.Targeted toxicants to dopaminergic neuronal cell death.Environmental neurotoxicant manganese regulates exosome-mediated extracellular miRNAs in cell culture model of Parkinson's disease: Relevance to α-synuclein misfolding in metal neurotoxicity.Fyn kinase regulates misfolded α-synuclein uptake and NLRP3 inflammasome activation in microgliaManganese activates NLRP3 inflammasome signaling and propagates exosomal release of ASC in microglial cellsManganese promotes the aggregation and prion-like cell-to-cell exosomal transmission of α-synuclein
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Dilshan S Harischandra
@ast
Dilshan S Harischandra
@en
Dilshan S Harischandra
@es
Dilshan S Harischandra
@nl
type
label
Dilshan S Harischandra
@ast
Dilshan S Harischandra
@en
Dilshan S Harischandra
@es
Dilshan S Harischandra
@nl
altLabel
Dilshan Harischandra
@en
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Dilshan S Harischandra
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Dilshan S Harischandra
@en
Dilshan S Harischandra
@es
Dilshan S Harischandra
@nl
P106
P1153
55382798600
P2798
P31
P496
0000-0003-3220-8765