about
Mitochondria and cardiovascular agingQuality control systems in cardiac agingAltered proteome turnover and remodeling by short-term caloric restriction or rapamycin rejuvenate the aging heart.Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.Sildenafil reverses cardiac dysfunction in the mdx mouse model of Duchenne muscular dystrophy.Age-dependent cardiomyopathy in mitochondrial mutator mice is attenuated by overexpression of catalase targeted to mitochondria.Respiratory chain protein turnover rates in mice are highly heterogeneous but strikingly conserved across tissues, ages, and treatments.Cardiac aging: from molecular mechanisms to significance in human health and diseaseSubacute calorie restriction and rapamycin discordantly alter mouse liver proteome homeostasis and reverse aging effectsHuman pluripotent stem cells: Prospects and challenges as a source of cardiomyocytes for in vitro modeling and cell-based cardiac repair.Genotype-phenotype correlation of maternally inherited disorders due to mutations in mitochondrial DNA.Rapamycin reverses elevated mTORC1 signaling in lamin A/C-deficient mice, rescues cardiac and skeletal muscle function, and extends survival.Mitochondrial targeted antioxidant Peptide ameliorates hypertensive cardiomyopathyMitochondrial oxidative stress mediates angiotensin II-induced cardiac hypertrophy and Galphaq overexpression-induced heart failureGlobal proteomics and pathway analysis of pressure-overload-induced heart failure and its attenuation by mitochondrial-targeted peptides.Loss of β-adrenergic-stimulated phosphorylation of CaV1.2 channels on Ser1700 leads to heart failureMitochondrial-Targeted Catalase: Extended Longevity and the Roles in Various Disease Models.Mitochondrial-targeted catalase is good for the old mouse proteome, but not for the young: 'reverse' antagonistic pleiotropy?Mitochondrial Maturation in Human Pluripotent Stem Cell Derived Cardiomyocytes.Effect of physical activity on the prevalence of metabolic syndrome and left ventricular hypertrophy in apparently healthy adults.Stable Isotope Labeling Reveals Novel Insights Into Ubiquitin-Mediated Protein Aggregation With Age, Calorie Restriction, and Rapamycin Treatment.DnaJ Homolog Subfamily B Member 9 Is a Putative Autoantigen in Fibrillary GN.Joint effects of N-terminal pro-B-type-natriuretic peptide and C-reactive protein vs angiographic severity in predicting major adverse cardiovascular events and clinical restenosis after coronary angioplasty in patients with stable coronary artery dInterstitial eosinophilic aggregates in diabetic nephropathy: allergy or not?Using structural equation model to illustrate the relationship between metabolic risk factors and cardiovascular complications in Taiwan.Localization and characterization of a novel secreted protein SCUBE1 in human platelets.Congestive hepatic fibrosis score: a novel histologic assessment of clinical severityPlasma Concentration of SCUBE1, a Novel Platelet Protein, Is Elevated in Patients With Acute Coronary Syndrome and Ischemic StrokeExploring the Effects of C-Reactive Protein (CRP) and Interleukin-1 β Single Nucleotide Polymorphisms on CRP Concentration in Patients With Established Coronary Artery DiseaseHuman C-reactive Protein (CRP) Gene 1059 G > C Polymorphism is Associated with Plasma CRP Concentration in Patients Receiving Coronary AngiographyThe Effects of Metabolic SyndromeVersusInfectious Burden on Inflammation, Severity of Coronary Atherosclerosis, and Major Adverse Cardiovascular EventsEpithelial innate immunity mediates tubular cell senescence after kidney injuryDifferential effects of various genetic mouse models of the mechanistic target of rapamycin complex I inhibition on heart failureMitotic Catastrophe Causes Podocyte Loss in the Urine of Human DiabeticsC3(H2O) prevents rescue of complement-mediated C3 glomerulopathy in Cfh-/- Cfd-/- miceGlutathione peroxidase-1 overexpression reduces oxidative stress, and improves pathology and proteome remodeling in the kidneys of old mice
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0001-7724-321X
@en
name
Dao-Fu Dai
@ast
Dao-Fu Dai
@en
Dao-Fu Dai
@es
Dao-Fu Dai
@nl
type
label
Dao-Fu Dai
@ast
Dao-Fu Dai
@en
Dao-Fu Dai
@es
Dao-Fu Dai
@nl
prefLabel
Dao-Fu Dai
@ast
Dao-Fu Dai
@en
Dao-Fu Dai
@es
Dao-Fu Dai
@nl
P106
P1153
14055667100
P31
P496
0000-0001-7724-321X