Altered proteome turnover and remodeling by short-term caloric restriction or rapamycin rejuvenate the aging heart. - Test2 - elhamkhani - TriplyDB

Altered proteome turnover and remodeling by short-term caloric restriction or rapamycin rejuvenate the aging heart.

Altered proteome turnover and remodeling by short-term caloric restriction or rapamycin rejuvenate the aging heart.

http://www.wikidata.org/entity/Q33692414

about

Geroscience approaches to increase healthspan and slow aging Inhibition of the Mechanistic Target of Rapamycin (mTOR)-Rapamycin and Beyond Healthy aging: The ultimate preventative medicine Biochemical Genetic Pathways that Modulate Aging in Multiple Species New insights into the role of mitochondrial dynamics and autophagy during oxidative stress and aging in the heart Autophagy in cardiovascular biology Cardiovascular proteomics in the era of big data: experimental and computational advances Quality control systems in cardiac aging Sex- and tissue-specific changes in mTOR signaling with age in C57BL/6J mice Intermittent Administration of Rapamycin Extends the Life Span of Female C57BL/6J Mice The Mechanistic Target of Rapamycin: The Grand ConducTOR of Metabolism and Aging Mitochondrial Metabolism in Aging Heart Mitochondrial dysfunction in cardiac aging Mitochondria and oxidative stress in heart aging Staying young at heart: autophagy and adaptation to cardiac aging Nuclear DNA damage signalling to mitochondria in ageing Dynamic regulation of genetic pathways and targets during aging in Caenorhabditis elegans.Moderate lifelong overexpression of tuberous sclerosis complex 1 (TSC1) improves health and survival in mice Globally Optimized Targeted Mass Spectrometry: Reliable Metabolomics Analysis with Broad Coverage On the Relationship between Energy Metabolism, Proteostasis, Aging and Parkinson's Disease: Possible Causative Role of Methylglyoxal and Alleviative Potential of Carnosine.Mechanisms of In Vivo Ribosome Maintenance Change in Response to Nutrient Signals.Functions of autophagy in pathological cardiac hypertrophy Respiratory chain protein turnover rates in mice are highly heterogeneous but strikingly conserved across tissues, ages, and treatments.Subacute calorie restriction and rapamycin discordantly alter mouse liver proteome homeostasis and reverse aging effects Targeted plasma metabolome response to variations in dietary glycemic load in a randomized, controlled, crossover feeding trial in healthy adults Systems biology approaches to study the molecular effects of caloric restriction and polyphenols on aging processes.Age-related loss of hepatic Nrf2 protein homeostasis: Potential role for heightened expression of miR-146a.The histone deacetylase inhibitor butyrate improves metabolism and reduces muscle atrophy during aging.Age modifies respiratory complex I and protein homeostasis in a muscle type-specific manner.Mitochondrial protein turnover: methods to measure turnover rates on a large scale.Rapamycin transiently induces mitochondrial remodeling to reprogram energy metabolism in old hearts.The Biology of Aging: Citizen Scientists and Their Pets as a Bridge Between Research on Model Organisms and Human Subjects.Age- and Hypertension-Associated Protein Aggregates in Mouse Heart Have Similar Proteomic Profiles.Aging and Autophagy in the Heart Adaptations to chronic rapamycin in mice.Pharmacological Strategies to Retard Cardiovascular Aging Rapamycin: An InhibiTOR of Aging Emerges From the Soil of Easter Island.Transient rapamycin treatment can increase lifespan and healthspan in middle-aged mice.Protein turnover analysis in Salmonella Typhimurium during infection by dynamic SILAC, Topograph, and quantitative proteomics.The Aging Heart

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P2860

Altered proteome turnover and remodeling by short-term caloric restriction or rapamycin rejuvenate the aging heart.

http://www.wikidata.org/entity/Q33692414

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2014 nî lūn-bûn

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2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Altered proteome turnover and ...... in rejuvenate the aging heart.

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Altered proteome turnover and ...... in rejuvenate the aging heart.

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Daniel Raftery

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Danijel Djukovic

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David Crispin

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Edward J Hsieh

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Haiwei Gu

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Michael J MacCoss

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Pabalu P Karunadharma

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Richard P Beyer

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Tony Chen

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Ying A Chiao

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P2860

Rapamycin, but not resveratrol or simvastatin, extends life span of genetically heterogeneous mice

Rapamycin-induced insulin resistance is mediated by mTORC2 loss and uncoupled from longevity

Aging as an event of proteostasis collapse

mTOR signaling in growth control and disease

Rapamycin fed late in life extends lifespan in genetically heterogeneous mice

Caloric restriction delays disease onset and mortality in rhesus monkeys

Defining the role of mTOR in cancer

Rapamycin-mediated inhibition of mammalian target of rapamycin in skeletal muscle cells reduces glucose utilization and increases fatty acid oxidation

The genetics of ageing

Regulation of the mTOR complex 1 pathway by nutrients, growth factors, and stress

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10.1111/ACEL.12203

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3

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13

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P S Rabinovitch

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