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Extracellular high-mobility group box 1 protein (HMGB1) as a mediator of persistent painInterleukin-6 secretion by astrocytes is dynamically regulated by PI3K-mTOR-calcium signalingCross-centre replication of suppressed burrowing behaviour as an ethologically relevant pain outcome measure in the rat: a prospective multicentre study.Tumor necrosis factor-alpha induces mechanical allodynia after spinal nerve ligation by activation of p38 MAPK in primary sensory neuronsEnsuring transparency and minimization of methodologic bias in preclinical pain research: PPRECISE considerationsGadd45beta deficiency in rheumatoid arthritis: enhanced synovitis through JNK signaling.Peripheral inflammation induces tumor necrosis factor dependent AMPA receptor trafficking and Akt phosphorylation in spinal cord in addition to pain behaviorLeader arrangement in the adenovirus fiber mRNA.Characterization of a low-molecular-weight virus-associated (VA) RNA encoded by simian adenovirus type 7 which functionally can substitute for adenovirus type 5 VA RNAICharacterization of the acute and persistent pain state present in K/BxN serum transfer arthritis.Spinal p38 MAP kinase is necessary for NMDA-induced spinal PGE(2) release and thermal hyperalgesia.Prostaglandin E2 release evoked by intrathecal dynorphin is dependent on spinal p38 mitogen activated protein kinase.Spinal actions of lipoxin A4 and 17(R)-resolvin D1 attenuate inflammation-induced mechanical hypersensitivity and spinal TNF release.Spinal phospholipase A2 in inflammatory hyperalgesia: role of group IVA cPLA2Increased levels of IL-6 in the cerebrospinal fluid of patients with chronic schizophrenia--significance for activation of the kynurenine pathway.Defective RNA splicing in the absence of adenovirus-associated RNAI.Spinal TLR4 mediates the transition to a persistent mechanical hypersensitivity after the resolution of inflammation in serum-transferred arthritis.Dietary nitrate improves cardiac contractility via enhanced cellular Ca²⁺ signaling.Peripheral inflammatory disease associated with centrally activated IL-1 system in humans and miceDelayed Imatinib Treatment for Acute Spinal Cord Injury: Functional Recovery and Serum BiomarkersIdentification of a novel chemokine-dependent molecular mechanism underlying rheumatoid arthritis-associated autoantibody-mediated bone loss.A mechanism by which adenovirus virus-associated RNAI controls translation in a transient expression assay.Inhibition of spinal cytosolic phospholipase A(2) expression by an antisense oligonucleotide attenuates tissue injury-induced hyperalgesia.Rat substrains differ in the magnitude of spontaneous locomotor recovery and in the development of mechanical hypersensitivity after experimental spinal cord injury.Spinal astrocytes in pain processing: non-neuronal cells as therapeutic targets.Inflammatory hyperalgesia induces essential bioactive lipid production in the spinal cord.Role of extracellular damage-associated molecular pattern molecules (DAMPs) as mediators of persistent pain.Pain in rheumatoid arthritis: models and mechanisms.Inflammation-induced GluA1 trafficking and membrane insertion of Ca2+ permeable AMPA receptors in dorsal horn neurons is dependent on spinal tumor necrosis factor, PI3 kinase and protein kinase A.Pattern recognition receptors in chronic pain: Mechanisms and therapeutic implications.Mechanisms leading from systemic autoimmunity to joint-specific disease in rheumatoid arthritis.Autoantibodies to citrullinated proteins induce joint pain independent of inflammation via a chemokine-dependent mechanism.Role of p38 mitogen activated protein kinase in a model of osteosarcoma-induced pain.The DNA binding domains of the yeast Gal4 and human c-Jun transcription factors interact through the zinc-finger and bZIP motifs.An adenovirus E1A transcriptional repressor domain functions as an activator when tethered to a promoterRole of spinal p38alpha and beta MAPK in inflammatory hyperalgesia and spinal COX-2 expression.Using gait analysis to assess weight bearing in rats with Freund׳s complete adjuvant-induced monoarthritis to improve predictivity: Interfering with the cyclooxygenase and nerve growth factor pathways.Adenovirus VA RNAI mediates a translational stimulation which is not restricted to the viral mRNAsPhenotypic changes in dorsal root ganglion and spinal cord in the collagen antibody-induced arthritis mouse model.The carboxy-terminal exon of the adenovirus E1A protein is required for E4F-dependent transcription activation.
P50
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Zweeds onderzoekster
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researcher ORCID ID = 0000-0001-7980-4631
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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Camilla I Svensson
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0000-0001-7980-4631
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