about
Leveraging two-way probe-level block design for identifying differential gene expression with high-density oligonucleotide arraysThe selection and function of cell type-specific enhancersCooperative NCoR/SMRT interactions establish a corepressor-based strategy for integration of inflammatory and anti-inflammatory signaling pathwaysSenataxin suppresses the antiviral transcriptional response and controls viral biogenesis53BP1 and USP28 mediate p53 activation and G1 arrest after centrosome loss or extended mitotic duration.In silico discovery of transcription regulatory elements in Plasmodium falciparum.The transcriptional landscape of mouse beta cells compared to human beta cells reveals notable species differences in long non-coding RNA and protein-coding gene expression.An alternative pluripotent state confers interspecies chimaeric competencyTargeted gene correction minimally impacts whole-genome mutational load in human-disease-specific induced pluripotent stem cell clones.Mechanisms establishing TLR4-responsive activation states of inflammatory response genes.Function of a Foxp3 cis-element in protecting regulatory T cell identityReprogramming transcription by distinct classes of enhancers functionally defined by eRNAControl of VEGF-A transcriptional programs by pausing and genomic compartmentalizationPrimate-specific ORF0 contributes to retrotransposon-mediated diversity.An evolutionarily conserved DNA architecture determines target specificity of the TWIST family bHLH transcription factors.Identification of novel long noncoding RNAs underlying vertebrate cardiovascular development.Sensitive ChIP-DSL technology reveals an extensive estrogen receptor alpha-binding program on human gene promoters.The DAXX co-repressor is directly recruited to active regulatory elements genome-wide to regulate autophagy programs in a model of human prostate cancer.Targeting the spliceosome in chronic lymphocytic leukemia with the macrolides FD-895 and pladienolide-B.The nucleoporin Nup153 regulates embryonic stem cell pluripotency through gene silencingMyofibroblasts revert to an inactive phenotype during regression of liver fibrosisNew roles for DNA cytosine modification, eRNA, anchors, and superanchors in developing B cell progenitors.BRD4 is a novel therapeutic target for liver fibrosis.Positive intergenic feedback circuitry, involving EBF1 and FOXO1, orchestrates B-cell fateSystems biology of macrophages.Evolution of a transcriptional regulator from a transmembrane nucleoporinEmergence of a Homo sapiens-specific gene family and chromosome 16p11.2 CNV susceptibilityRev-Erbs repress macrophage gene expression by inhibiting enhancer-directed transcription.Presenilins regulate neurotrypsin gene expression and neurotrypsin-dependent agrin cleavage via cyclic AMP response element-binding protein (CREB) modulationNascent RNA sequencing reveals distinct features in plant transcription.Nucleoporin-mediated regulation of cell identity genes.Topoisomerase 1 inhibition suppresses inflammatory genes and protects from death by inflammation.Mesothelin/mucin 16 signaling in activated portal fibroblasts regulates cholestatic liver fibrosis.Multilineage priming of enhancer repertoires precedes commitment to the B and myeloid cell lineages in hematopoietic progenitors.Effect of natural genetic variation on enhancer selection and function.Differential L1 regulation in pluripotent stem cells of humans and apes.Bcl-6 and NF-kappaB cistromes mediate opposing regulation of the innate immune response.Molecular determinants of crosstalk between nuclear receptors and toll-like receptors.Mechanisms of in vivo binding site selection of the hematopoietic master transcription factor PU.1.SMADs and YAP compete to control elongation of β-catenin:LEF-1-recruited RNAPII during hESC differentiation.
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P50
description
investigador
@es
researcher
@en
name
Chris Benner
@en
Chris Benner
@nl
type
label
Chris Benner
@en
Chris Benner
@nl
prefLabel
Chris Benner
@en
Chris Benner
@nl
P31
P3762
P496
0000-0002-4618-0719