S100A14 stimulates cell proliferation and induces cell apoptosis at different concentrations via receptor for advanced glycation end products (RAGE)
about
Therapeutic targeting of tumor growth and angiogenesis with a novel anti-S100A4 monoclonal antibodySolution structure and dynamics of human S100A14The multiple faces of RAGE--opportunities for therapeutic intervention in aging and chronic diseaseCo-expression of S100A14 and S100A16 correlates with a poor prognosis in human breast cancer and promotes cancer cell invasion.S100P antibody-mediated therapy as a new promising strategy for the treatment of pancreatic cancer.Exendin-4, a glucagon-like peptide-1 receptor agonist, inhibits hyperglycemia-induced apoptosis in myocytes by suppressing receptor for advanced glycation end products expressionProteomic plasma membrane profiling reveals an essential role for gp96 in the cell surface expression of LDLR family members, including the LDL receptor and LRP6.Hornerin, an S100 family protein, is functional in breast cells and aberrantly expressed in breast cancerCell migration is regulated by AGE-RAGE interaction in human oral cancer cells in vitro.S100A14 interacts with S100A16 and regulates its expression in human cancer cells.S100A14, a mediator of epithelial-mesenchymal transition, regulates proliferation, migration and invasion of human cervical cancer cells.Involvement of S100A14 protein in cell invasion by affecting expression and function of matrix metalloproteinase (MMP)-2 via p53-dependent transcriptional regulationRAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.Overexpression of S100A14 in human serous ovarian carcinomaFunctions of S100 proteins.S100A14, a member of the EF-hand calcium-binding proteins, is overexpressed in breast cancer and acts as a modulator of HER2 signaling.Advanced glycosylation end product promotes forkhead box O1 and inhibits Wnt pathway to suppress capacities of epidermal stem cells.S100 protein family in human cancer.12-O-tetradecanoylphorbol-13-acetate promotes breast cancer cell motility by increasing S100A14 level in a Kruppel-like transcription factor 4 (KLF4)-dependent manner.Receptor for advanced glycation end products (RAGE) and its ligands: focus on spinal cord injury.S100A6 promotes cell proliferation in human nasopharyngeal carcinoma via the p38/MAPK signaling pathway.Downregulation of 425G>a variant of calcium-binding protein S100A14 associated with poor differentiation and prognosis in gastric cancer.Correction: S100A14 Stimulates Cell Proliferation and Induces Cell Apoptosis at Different Concentrations via Receptor for Advanced Glycation End Products (RAGE).Soluble receptor for advanced glycation end products alleviates nephritis in (NZB/NZW)F1 mice.Receptor for advanced glycation end products contributes to postnatal pulmonary development and adult lung maintenance program in mice.S100A14 rs11548103 G>A polymorphism is associated with a decreased risk of esophageal cancer in a Chinese population.S100A6 promotes proliferation of intrahepatic cholangiocarcinoma cells via the activation of the p38/MAPK pathway.S100A7: from mechanism to cancer therapy.Up-regulation of receptors for advanced glycation end-products by alveolar epithelium influences cytodifferentiation and causes severe lung hypoplasia.In vitro anticancer effects of a RAGE inhibitor discovered using a structure-based drug design system.Loss of S100A14 expression is associated with the progression of adenocarcinomas of the small intestine.Therapeutic positioning of secretory acetylated APE1/Ref-1 requirement for suppression of tumor growth in triple-negative breast cancer in vivo.Overexpression of S100A14 contributes to malignant progression and predicts poor prognosis of lung adenocarcinoma.S100A8/A9 promotes parenchymal damage and renal fibrosis in obstructive nephropathy
P2860
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P2860
S100A14 stimulates cell proliferation and induces cell apoptosis at different concentrations via receptor for advanced glycation end products (RAGE)
description
2011 nî lūn-bûn
@nan
2011 թուականին հրատարակուած գիտական յօդուած
@hyw
2011 թվականին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@ast
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@en
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@en-gb
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@nl
type
label
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@ast
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@en
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@en-gb
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@nl
prefLabel
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@ast
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@en
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@en-gb
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@nl
P2093
P2860
P1433
P1476
S100A14 stimulates cell prolif ...... glycation end products (RAGE)
@en
P2093
Aiping Luo
Hongyan Chen
Qing'e Jin
Zhihua Liu
P2860
P304
P356
10.1371/JOURNAL.PONE.0019375
P407
P577
2011-01-01T00:00:00Z