RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.
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Plausible Roles for RAGE in Conditions Exacerbated by Direct and Indirect (Secondhand) Smoke Exposure.Sex-specific lung functional changes in adult mice exposed only to second-hand smoke in utero.Antenatal exposure of maternal secondhand smoke (SHS) increases fetal lung expression of RAGE and induces RAGE-mediated pulmonary inflammation.Nontypeable Haemophilus Influenzae Infection Upregulates the NLRP3 Inflammasome and Leads to Caspase-1-Dependent Secretion of Interleukin-1β - A Possible Pathway of Exacerbations in COPDEffect of hospital use of oral nutritional supplementation on length of stay, hospital cost, and 30-day readmissions among Medicare patients with COPD.Soft TCPTP Agonism-Novel Target to Rescue Airway Epithelial Integrity by Exogenous SpermidineAssociation of polymorphisms of the receptor for advanced glycation end products gene with COPD in the Chinese population.Blockade of RAGE ameliorates elastase-induced emphysema development and progression via RAGE-DAMP signaling.Second hand smoke and COPD: lessons from animal studies.Conditional over-expression of RAGE by embryonic alveolar epithelium compromises the respiratory membrane and impairs endothelial cell differentiation.Conditionally induced RAGE expression by proximal airway epithelial cells in transgenic mice causes lung inflammation.Inhibition of the receptor for advanced glycation end-products (RAGE) protects from secondhand smoke (SHS)-induced intrauterine growth restriction IUGR in mice.Effect of long-term maternal smoking on the offspring's lung health.Cigarette smoke extract induces oral squamous cell carcinoma cell invasion in a receptor for advanced glycation end-products-dependent manner.Cardiomyocyte mitochondrial respiration is reduced by receptor for advanced glycation end-product signaling in a ceramide-dependent manner.Acute secondhand smoke-induced pulmonary inflammation is diminished in RAGE knockout mice.COPD as an endothelial disorder: endothelial injury linking lesions in the lungs and other organs? (2017 Grover Conference Series).Receptor for advanced glycation end products and soluble receptor for advanced glycation end products: a balancing act in chronic obstructive pulmonary disease?
P2860
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P2860
RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.
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2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.
@ast
RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.
@en
type
label
RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.
@ast
RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.
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prefLabel
RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.
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RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.
@en
P2093
P2860
P356
P1476
RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.
@en
P2093
Adam B Robinson
Jeffrey A Stogsdill
Joshua B Lewis
Paul R Reynolds
Tyler T Wood
P2860
P356
10.3389/FPHYS.2012.00301
P577
2012-07-25T00:00:00Z