The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
about
The reticulons: a family of proteins with diverse functionsNogo limits neural plasticity and recovery from injuryNogo receptor inhibition enhances functional recovery following lysolecithin-induced demyelination in mouse optic chiasmAstrocytes in multiple sclerosis: a product of their environmentNogo-receptors NgR1 and NgR2 do not mediate regulation of CD4 T helper responses and CNS repair in experimental autoimmune encephalomyelitisSelective temporal and regional alterations of Nogo-A and small proline-rich repeat protein 1A (SPRR1A) but not Nogo-66 receptor (NgR) occur following traumatic brain injury in the ratA novel unbiased proteomic approach to detect the reactivity of cerebrospinal fluid in neurological diseasesEffects of Bu Shen Yi sui capsule on NogoA/NgR and its signaling pathways RhoA/ROCK in mice with experimental autoimmune encephalomyelitisCombination treatment of experimental stroke with Niaspan and Simvastatin, reduces axonal damage and improves functional outcomeDeveloping therapeutics for the treatment of multiple sclerosisNogo-A expression in the brain of mice with cerebral malariaNeuroprotective effect of combination therapy of glatiramer acetate and epigallocatechin-3-gallate in neuroinflammation.Amyotrophic lateral sclerosis: a focus on disease progressionSilencing Nogo-A promotes functional recovery in demyelinating disease.Niacin treatment of stroke increases synaptic plasticity and axon growth in rats.Angiogenesis in multiple sclerosis and experimental autoimmune encephalomyelitis.Overexpression of glia maturation factor reinstates susceptibility to myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitis in glia maturation factor deficient mice.Proteolytic control of neurite outgrowth inhibitor NOGO-A by the cAMP/PKA pathway.Nogo-receptor 1 deficiency has no influence on immune cell repertoire or function during experimental autoimmune encephalomyelitis.Emerging therapies for treatment of multiple sclerosisDiscovery, optimization, and pharmacological characterization of novel heteroaroylphenylureas antagonists of C-C chemokine ligand 2 functionThe axon-glia unit in white matter stroke: mechanisms of damage and recovery.Thymic gene transfer of myelin oligodendrocyte glycoprotein ameliorates the onset but not the progression of autoimmune demyelination.Early intervention with gene-modified mesenchymal stem cells overexpressing interleukin-4 enhances anti-inflammatory responses and functional recovery in experimental autoimmune demyelination.Therapeutic benefit of treatment of stroke with simvastatin and human umbilical cord blood cells: neurogenesis, synaptic plasticity, and axon growthThe fibrin-derived gamma377-395 peptide inhibits microglia activation and suppresses relapsing paralysis in central nervous system autoimmune disease.Therapeutic vaccination for central nervous system repair.Toward biomarkers in multiple sclerosis: new advances.Limiting multiple sclerosis related axonopathy by blocking Nogo receptor and CRMP-2 phosphorylation.Diabetes-induced central neuritic dystrophy and cognitive deficits are associated with the formation of oligomeric reticulon-3 via oxidative stress.Delayed administration of a PTEN inhibitor BPV improves functional recovery after experimental strokePhosphorylation and compactness of neurofilaments in multiple sclerosis: indicators of axonal pathology.Nogo-A and Nogo-66 receptor in amyotrophic lateral sclerosis.MitoQ, a mitochondria-targeted antioxidant, delays disease progression and alleviates pathogenesis in an experimental autoimmune encephalomyelitis mouse model of multiple sclerosis."Opening" the mesenchymal stem cell tool box.Axonal degeneration as a therapeutic target in the CNS.Multiple sclerosis: does Nogo play a role?New findings and old controversies in the research of multiple sclerosis and its model experimental autoimmune encephalomyelitis.The role of reticulons in neurodegenerative diseases.CRMP2: functional roles in neural development and therapeutic potential in neurological diseases.
P2860
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P2860
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
description
2004 nî lūn-bûn
@nan
2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
@ast
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
@en
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
@nl
type
label
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
@ast
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
@en
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
@nl
prefLabel
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
@ast
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
@en
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
@nl
P2093
P2860
P356
P1433
P1476
The neurite outgrowth inhibitor Nogo A is involved in autoimmune-mediated demyelination
@en
P2093
Belinda M Murray
Ben Barres
Binhai Zheng
Claude C A Bernard
Jonathan L McQualter
Kelly A Jordan
Peggy P Ho
Tara Karnezis
Wim Mandemakers
P2860
P2888
P304
P356
10.1038/NN1261
P407
P577
2004-07-01T00:00:00Z