MitoQ, a mitochondria-targeted antioxidant, delays disease progression and alleviates pathogenesis in an experimental autoimmune encephalomyelitis mouse model of multiple sclerosis.
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The Therapeutic Potential of the Ketogenic Diet in Treating Progressive Multiple SclerosisInflammation, Iron, Energy Failure, and Oxidative Stress in the Pathogenesis of Multiple SclerosisNeuroprotective properties of the marine carotenoid astaxanthin and omega-3 fatty acids, and perspectives for the natural combination of both in krill oilEffects of coenzyme Q10 on the ratio of TH1/TH2 in experimental autoimmune encephalomyelitis model of multiple sclerosis in C57BL/6.Mitochondria-derived reactive oxygen species mediate caspase-dependent and -independent neuronal deaths.Mitochondria-targeting particles.Deletion of mitochondrial anchoring protects dysmyelinating shiverer: implications for progressive MS.Excitotoxins, Mitochondrial and Redox Disturbances in Multiple Sclerosis.Mitochondria-targeted molecules MitoQ and SS31 reduce mutant huntingtin-induced mitochondrial toxicity and synaptic damage in Huntington's disease.Caged Garcinia Xanthones, a Novel Chemical Scaffold with Potent Antimalarial ActivityGarrison Institute on Aging: A New Hope for Elderly Individuals and Patients with Alzheimer's Disease.Antioxidative defense mechanisms controlled by Nrf2: state-of-the-art and clinical perspectives in neurodegenerative diseases.Effects of transcranial magnetic stimulation on oxidative stress in experimental autoimmune encephalomyelitis.Mitochondrial dysfunction and oxidative stress in metabolic disorders - A step towards mitochondria based therapeutic strategies.Modulating inflammation and neuroprotection in multiple sclerosis.Improvement of oxidative and metabolic parameters by cellfood administration in patients affected by neurodegenerative diseases on chelation treatment.Metformin Inhibits the Production of Reactive Oxygen Species from NADH:Ubiquinone Oxidoreductase to Limit Induction of Interleukin-1β (IL-1β) and Boosts Interleukin-10 (IL-10) in Lipopolysaccharide (LPS)-activated Macrophages.Oxidative tissue injury in multiple sclerosis is only partly reflected in experimental disease models.Multiple Immune-Inflammatory and Oxidative and Nitrosative Stress Pathways Explain the Frequent Presence of Depression in Multiple Sclerosis.NLR-Dependent Regulation of Inflammation in Multiple Sclerosis- Dependent Regulation of Inflammation in Multiple Sclerosis.Efficacy and Tolerability of Phytomedicines in Multiple Sclerosis Patients: A Review.
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MitoQ, a mitochondria-targeted antioxidant, delays disease progression and alleviates pathogenesis in an experimental autoimmune encephalomyelitis mouse model of multiple sclerosis.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 19 September 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
MitoQ, a mitochondria-targeted ...... e model of multiple sclerosis.
@en
MitoQ, a mitochondria-targeted ...... e model of multiple sclerosis.
@nl
type
label
MitoQ, a mitochondria-targeted ...... e model of multiple sclerosis.
@en
MitoQ, a mitochondria-targeted ...... e model of multiple sclerosis.
@nl
prefLabel
MitoQ, a mitochondria-targeted ...... e model of multiple sclerosis.
@en
MitoQ, a mitochondria-targeted ...... e model of multiple sclerosis.
@nl
P2093
P2860
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MitoQ, a mitochondria-targeted ...... e model of multiple sclerosis.
@en
P2093
Maria Manczak
P Hemachandra Reddy
Peizhong Mao
Ulziibat P Shirendeb
P2860
P304
P356
10.1016/J.BBADIS.2013.09.005
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P577
2013-09-19T00:00:00Z