TASK1 modulates inflammation and neurodegeneration in autoimmune inflammation of the central nervous system. - Wikidata - thesis-toulmin - TriplyDB

TASK1 modulates inflammation and neurodegeneration in autoimmune inflammation of the central nervous system.

TASK1 modulates inflammation and neurodegeneration in autoimmune inflammation of the central nervous system.

http://www.wikidata.org/entity/Q30433284

about

Pathophysiological processes in multiple sclerosis: focus on nuclear factor erythroid-2-related factor 2 and emerging pathways Mechanisms of oxidative damage in multiple sclerosis and neurodegenerative diseases: therapeutic modulation via fumaric acid esters A human TREK-1/HEK cell line: a highly efficient screening tool for drug development in neurological diseases Potent and selective inhibitors of the TASK-1 potassium channel through chemical optimization of a bis-amide scaffold.Nicotinic acid adenine dinucleotide phosphate-mediated calcium signalling in effector T cells regulates autoimmunity of the central nervous system.Cooperative endocytosis of the endosomal SNARE protein syntaxin-8 and the potassium channel TASK-1.The endocannabinoid system as a target for the treatment of neurodegenerative disease.TRPM2 cation channels modulate T cell effector functions and contribute to autoimmune CNS inflammation.Fumaric acid esters exert neuroprotective effects in neuroinflammation via activation of the Nrf2 antioxidant pathway.Regulation of neurovascular coupling in autoimmunity to water and ion channels Expression of K2P5.1 potassium channels on CD4+ T lymphocytes correlates with disease activity in rheumatoid arthritis patients TASK-1 Regulates Apoptosis and Proliferation in a Subset of Non-Small Cell Lung Cancers.Murine K2P5.1 Deficiency Has No Impact on Autoimmune Neuroinflammation due to Compensatory K2P3.1- and KV1.3-Dependent Mechanisms Ion channels in innate and adaptive immunity Ion channels in autoimmune neurodegeneration.Mechanisms of neurodegeneration and axonal dysfunction in multiple sclerosis.The CNS under pathophysiologic attack--examining the role of K₂p channels.The family of K2P channels: salient structural and functional properties.Much more than a leak: structure and function of K₂p-channels.Enhancement of TWIK-related acid-sensitive potassium channel 3 (TASK3) two-pore domain potassium channel activity by tumor necrosis factor α.Targeting ion channels for the treatment of autoimmune neuroinflammation.Functional and molecular identification of a TASK-1 potassium channel regulating chloride secretion through CFTR channels in the shark rectal gland: implications for cystic fibrosis.Expression of TASK-2 and its upregulation by B cell receptor stimulation in WEHI-231 mouse immature B cells.TASK-1 channels in oligodendrocytes: a role in ischemia mediated disruption The Effect of Mechanical Ventilation on TASK-1 Expression in the Brain in a Rat Model.Phospholipase D1 mediates lymphocyte adhesion and migration in experimental autoimmune encephalomyelitis.Presence of task-1 channel in the laryngeal mucosa in the newborn lamb.

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TASK1 modulates inflammation and neurodegeneration in autoimmune inflammation of the central nervous system.

http://www.wikidata.org/entity/Q30433284

description

2009 nî lūn-bûn

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2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հուլիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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TASK1 modulates inflammation a ...... of the central nervous system.

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Alexander M Herrmann

http://www.w3.org/2001/XMLSchema#string

Andreas Weishaupt

http://www.w3.org/2001/XMLSchema#string

Kerstin Göbel

http://www.w3.org/2001/XMLSchema#string

Nico Melzer

http://www.w3.org/2001/XMLSchema#string

Ole J Simon

http://www.w3.org/2001/XMLSchema#string

Stefan Bittner

http://www.w3.org/2001/XMLSchema#string

Sven G Meuth

http://www.w3.org/2001/XMLSchema#string

Thomas Budde

http://www.w3.org/2001/XMLSchema#string

P2860

The voltage-gated Kv1.3 K(+) channel in effector memory T cells as new target for MS

Endocannabinoids control spasticity in a multiple sclerosis model

Potassium leak channels and the KCNK family of two-P-domain subunits

The immunopathology of multiple sclerosis: an overview

The endocannabinoid anandamide is a direct and selective blocker of the background K(+) channel TASK-1.

Disruption of Cnp1 uncouples oligodendroglial functions in axonal support and myelination

Isolation and structure of a brain constituent that binds to the cannabinoid receptor

Central memory and effector memory T cell subsets: function, generation, and maintenance

Axonal transection in the lesions of multiple sclerosis

The TASK family: two-pore domain background K+ channels.

P304

2501-2516

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scholarly article

P356

10.1093/BRAIN/AWP163

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P407

P433

Pt 9

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P478

132

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Martin Bendszus

Douglas A Bayliss

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2009-07-01T00:00:00Z

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26334465

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19570851

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3031313

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