CREB binding protein is required for both short-term and long-term memory formation. - Wikidata - thesis-toulmin - TriplyDB

CREB binding protein is required for both short-term and long-term memory formation.

CREB binding protein is required for both short-term and long-term memory formation.

http://www.wikidata.org/entity/Q30476167

about

The endocannabinoid system in normal and pathological brain ageing Epigenetics in the mature mammalian brain: effects on behavior and synaptic transmission Hippocampal Transcriptome Profile of Persistent Memory Rescue in a Mouse Model of THRA1 Mutation-Mediated Resistance to Thyroid Hormone.Opposite monosynaptic scaling of BLP-vCA1 inputs governs hopefulness- and helplessness-modulated spatial learning and memory Epigenetic Changes in Neurodegenerative Diseases Epigenetic Modifications, Alcoholic Brain and Potential Drug Targets Prefrontal consolidation supports the attainment of fear memory accuracy Basal variability in CREB phosphorylation predicts trait-like differences in amygdala-dependent memory The role of nuclear PKMζ in memory maintenance.CBP is required for environmental enrichment-induced neurogenesis and cognitive enhancement.Epigenetic regulation of estrogen-dependent memory.The role of histone acetylation in memory formation and cognitive impairments.Epigenetic memory: the Lamarckian brain K-Lysine acetyltransferase 2a regulates a hippocampal gene expression network linked to memory formation CREB-binding protein levels in the rat hippocampus fail to predict chronological or cognitive aging Acetylation-mediated suppression of transcription-independent memory: bidirectional modulation of memory by acetylation Epigenetic mechanisms in fear conditioning: implications for treating post-traumatic stress disorder.Regular moderate or intense exercise prevents depression-like behavior without change of hippocampal tryptophan content in chronically tryptophan-deficient and stressed mice Hippocampal focal knockout of CBP affects specific histone modifications, long-term potentiation, and long-term memory.Histone acetylation and CREB binding protein are required for neuronal resistance against ischemic injury.Minocycline reduces neuroinflammation but does not ameliorate neuron loss in a mouse model of neurodegeneration Borna disease virus phosphoprotein modulates epigenetic signaling in neurons to control viral replication Epigenetic mechanisms in memory and synaptic function.Conditional deletion of Notch1 and Notch2 genes in excitatory neurons of postnatal forebrain does not cause neurodegeneration or reduction of Notch mRNAs and proteins The effects of perinatal choline supplementation on hippocampal cholinergic development in rats exposed to alcohol during the brain growth spurt Acetyltransferases (HATs) as targets for neurological therapeutics Epigenetics-beyond the genome in alcoholism.Xylaria nigripes mitigates spatial memory impairment induced by rapid eye movement sleep deprivation Genetic polymorphism of nonsyndromic cleft lip with or without cleft palate is associated with developmental dyslexia in Chinese school-aged populations.Transcriptional and epigenetic regulation of Hebbian and non-Hebbian plasticity.HDAC3 and the molecular brake pad hypothesis Epigenetic regulation of memory formation and maintenance.Lysine acetyltransferases CBP and p300 as therapeutic targets in cognitive and neurodegenerative disorders.Fibroblast growth factor-2 signaling in neurogenesis and neurodegeneration.Epigenetic regulation of memory by acetylation and methylation of chromatin: implications in neurological disorders, aging, and addiction.Nervous translation, do you get the message? A review of mRNPs, mRNA-protein interactions and translational control within cells of the nervous system.Histone-acetylation: a link between Alzheimer's disease and post-traumatic stress disorder?Cognitive dysfunction in Huntington's disease: mechanisms and therapeutic strategies beyond BDNF.Epigenetic regulation of cognition: A circumscribed review of the field.CBP-Dependent memory consolidation in the prefrontal cortex supports object-location learning.

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CREB binding protein is required for both short-term and long-term memory formation.

http://www.wikidata.org/entity/Q30476167

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

CREB binding protein is required for both short-term and long-term memory formation.

@ast

CREB binding protein is required for both short-term and long-term memory formation.

@en

type

label

CREB binding protein is required for both short-term and long-term memory formation.

@ast

CREB binding protein is required for both short-term and long-term memory formation.

@en

prefLabel

CREB binding protein is required for both short-term and long-term memory formation.

@ast

CREB binding protein is required for both short-term and long-term memory formation.

@en

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P356

JNEUROSCI.2378-10.2010

P1433

Journal of Neuroscience

P1476

CREB binding protein is required for both short-term and long-term memory formation.

@en

P2093

Guiquan Chen

http://www.w3.org/2001/XMLSchema#string

Hirotaka Watanabe

http://www.w3.org/2001/XMLSchema#string

Jan M van Deursen

http://www.w3.org/2001/XMLSchema#string

Jie Shen

http://www.w3.org/2001/XMLSchema#string

Xiaoyan Zou

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P2860

Enhanced long-term potentiation and impaired learning in mice with mutant postsynaptic density-95 protein

Presenilins are essential for regulating neurotransmitter release

A mouse model of Rubinstein-Taybi syndrome: defective long-term memory is ameliorated by inhibitors of phosphodiesterase 4

Conditional forebrain inactivation of nicastrin causes progressive memory impairment and age-related neurodegeneration

CBP/p300 in cell growth, transformation, and development

CBP histone acetyltransferase activity is a critical component of memory consolidation

Gene dosage-dependent embryonic development and proliferation defects in mice lacking the transcriptional integrator p300

Rubinstein-Taybi syndrome caused by mutations in the transcriptional co-activator CBP

The essential role of hippocampal CA1 NMDA receptor-dependent synaptic plasticity in spatial memory

Phosphorylation of the AMPA receptor GluR1 subunit is required for synaptic plasticity and retention of spatial memory

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10.1523/JNEUROSCI.2378-10.2010

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39

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2953943

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