The transcriptional regulators TAZ and YAP direct transforming growth factor β-induced tumorigenic phenotypes in breast cancer cells.
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Signaling in Fibrosis: TGF-β, WNT, and YAP/TAZ ConvergeA Review: Molecular Aberrations within Hippo Signaling in Bone and Soft-Tissue SarcomasCell density sensing alters TGF-β signaling in a cell-type-specific manner, independent from Hippo pathway activationTargeting the Hippo Signaling Pathway for Tissue Regeneration and Cancer TherapyYAP and TAZ: a nexus for Hippo signaling and beyondContext-dependent switch in chemo/mechanotransduction via multilevel crosstalk among cytoskeleton-regulated MRTF and TAZ and TGFβ-regulated Smad3Transforming growth factor-beta1 suppresses hepatocellular carcinoma proliferation via activation of Hippo signalingTranscriptional co-activator with PDZ-binding motif overexpression promotes cell proliferation and transcriptional co-activator with PDZ-binding motif deficiency induces cell cycle arrest in neuroblastoma.Concerted loss of TGFβ-mediated proliferation control and E-cadherin disrupts epithelial homeostasis and causes oral squamous cell carcinoma.A YAP/TAZ-Regulated Molecular Signature Is Associated with Oral Squamous Cell Carcinoma.Myocardin-related Transcription Factor Regulates Nox4 Protein Expression: LINKING CYTOSKELETAL ORGANIZATION TO REDOX STATE.Crumbs3-Mediated Polarity Directs Airway Epithelial Cell Fate through the Hippo Pathway Effector Yap.Accumulation of duct cells with activated YAP parallels fibrosis progression in non-alcoholic fatty liver disease.High expression of TAZ indicates a poor prognosis in retinoblastoma.Disruption of microRNA-21 by TALEN leads to diminished cell transformation and increased expression of cell-environment interaction genes.The interplay between TEAD4 and KLF5 promotes breast cancer partially through inhibiting the transcription of p27Kip1.Prognostic and clinicopathological role of long non-coding RNA UCA1 in various carcinomas.RUNX2 and TAZ-dependent signaling pathways regulate soluble E-Cadherin levels and tumorsphere formation in breast cancer cells.Long Noncoding RNA CUDR Regulates HULC and β-Catenin to Govern Human Liver Stem Cell Malignant Differentiation.EZH2 promotes progression of small cell lung cancer by suppressing the TGF-β-Smad-ASCL1 pathway.Hippo Signaling Mediators Yap and Taz Are Required in the Epicardium for Coronary Vasculature DevelopmentIdentification of chromatin accessibility domains in human breast cancer stem cells.Nuclear localization of TEF3-1 promotes cell cycle progression and angiogenesis in cancer.ω-3 PUFAs ameliorate liver fibrosis and inhibit hepatic stellate cells proliferation and activation by promoting YAP/TAZ degradation.TAZ promotes cell growth and inhibits Celastrol-induced cell apoptosis.Methylglyoxal, a glycolysis side-product, induces Hsp90 glycation and YAP-mediated tumor growth and metastasis.Epigenetic regulation of long noncoding RNA UCA1 by SATB1 in breast cancerThe Hippo signaling pathway provides novel anti-cancer drug targets.Hippo pathway in mammary gland development and breast cancer.The regulation and function of YAP transcription co-activator.ZEB1 turns into a transcriptional activator by interacting with YAP1 in aggressive cancer typesThe Hippo transducers TAZ and YAP in breast cancer: oncogenic activities and clinical implications.Urothelial cancer associated 1: a long noncoding RNA with a crucial role in cancer.Recent advances in understanding contextual TGFβ signalingFrom vestigial to vestigial-like: the Drosophila gene that has taken wing.The Hippo transducer TAZ promotes epithelial to mesenchymal transition and cancer stem cell maintenance in oral cancer.Arhgef7 promotes activation of the Hippo pathway core kinase LatsBiomechanical relationships between the corneal endothelium and Descemet's membrane.SMAD3 Activation: A Converging Point of Dysregulated TGF-Beta Superfamily Signaling and Genetic Aberrations in Granulosa Cell Tumor Development?Role of YAP/TAZ transcriptional regulators in resistance to anti-cancer therapies.
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P2860
The transcriptional regulators TAZ and YAP direct transforming growth factor β-induced tumorigenic phenotypes in breast cancer cells.
description
2014 nî lūn-bûn
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2014 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մարտին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
The transcriptional regulators ...... otypes in breast cancer cells.
@ast
The transcriptional regulators ...... otypes in breast cancer cells.
@en
type
label
The transcriptional regulators ...... otypes in breast cancer cells.
@ast
The transcriptional regulators ...... otypes in breast cancer cells.
@en
prefLabel
The transcriptional regulators ...... otypes in breast cancer cells.
@ast
The transcriptional regulators ...... otypes in breast cancer cells.
@en
P2860
P356
P1476
The transcriptional regulators ...... otypes in breast cancer cells.
@en
P2093
Samantha E Hiemer
P2860
P304
13461-13474
P356
10.1074/JBC.M113.529115
P407
P577
2014-03-19T00:00:00Z