Accumulation of duct cells with activated YAP parallels fibrosis progression in non-alcoholic fatty liver disease. - Wikidata - thesis-toulmin - TriplyDB

Accumulation of duct cells with activated YAP parallels fibrosis progression in non-alcoholic fatty liver disease.

Accumulation of duct cells with activated YAP parallels fibrosis progression in non-alcoholic fatty liver disease.

http://www.wikidata.org/entity/Q36075087

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Role of Hedgehog Signaling Pathway in NASH Molecular Pathogenesis of NASH Context-dependent switch in chemo/mechanotransduction via multilevel crosstalk among cytoskeleton-regulated MRTF and TAZ and TGFβ-regulated Smad3 Osteopontin is a proximal effector of leptin-mediated non-alcoholic steatohepatitis (NASH) fibrosis.ω-3 PUFAs ameliorate liver fibrosis and inhibit hepatic stellate cells proliferation and activation by promoting YAP/TAZ degradation.Nonalcoholic fatty liver disease: one entity, multiple impacts on liver health.Emerging concepts in biliary repair and fibrosis.Hepatic stellate cells as key target in liver fibrosis.Enhanced Steatosis and Fibrosis in Liver of Adult Offspring Exposed to Maternal High-Fat Diet Hepatocyte TAZ/WWTR1 Promotes Inflammation and Fibrosis in Nonalcoholic Steatohepatitis.STAT3 deficiency prevents hepatocarcinogenesis and promotes biliary proliferation in thioacetamide-induced liver injury.Oroxylin A inhibits ethanol-induced hepatocyte senescence via YAP pathway.Hedgehog signalling in liver pathophysiology.Pathogenesis of non-alcoholic fatty liver disease mediated by YAP.Hippo pathway coactivators Yap and Taz are required to coordinate mammalian liver regeneration.Furan-induced transcriptomic and gene-specific DNA methylation changes in the livers of Fischer 344 rats in a 2-year carcinogenicity study.The hedgehog pathway in nonalcoholic fatty liver disease.A Simple Diet- and Chemical-Induced Murine NASH Model with Rapid Progression of Steatohepatitis, Fibrosis and Liver Cancer.Pathogenesis of Nonalcoholic Steatohepatitis.

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P2860

Accumulation of duct cells with activated YAP parallels fibrosis progression in non-alcoholic fatty liver disease.

http://www.wikidata.org/entity/Q36075087

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2015 nî lūn-bûn

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2015年の論文

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Accumulation of duct cells wit ...... alcoholic fatty liver disease.

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Accumulation of duct cells wit ...... alcoholic fatty liver disease.

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Accumulation of duct cells wit ...... alcoholic fatty liver disease.

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Accumulation of duct cells wit ...... alcoholic fatty liver disease.

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J.JHEP.2015.05.031

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Journal of Hepatology

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Accumulation of duct cells wit ...... -alcoholic fatty liver disease

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Anna Mae Diehl

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Gregory Alexander Michelotti

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Guanhua Xie

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Helena Cortez-Pinto

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Mariana Verdelho Machado

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Thiago Almeida Pereira

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P2860

Regulation of the Hippo-YAP pathway by G-protein-coupled receptor signaling

Non-alcoholic fatty liver disease: what the clinician needs to know.

Osteopontin deletion prevents the development of obesity and hepatic steatosis via impaired adipose tissue matrix remodeling and reduced inflammation and fibrosis in adipose tissue and liver in mice

Third Report of the National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (Adult Treatment Panel III) final report

YAP1 is amplified and up-regulated in hedgehog-associated medulloblastomas and mediates Sonic hedgehog-driven neural precursor proliferation

YAP1 increases organ size and expands undifferentiated progenitor cells

Design and validation of a histological scoring system for nonalcoholic fatty liver disease

Reduced lipoapoptosis, hedgehog pathway activation and fibrosis in caspase-2 deficient mice with non-alcoholic steatohepatitis.

Mouse models of diet-induced nonalcoholic steatohepatitis reproduce the heterogeneity of the human disease.

The transcriptional regulators TAZ and YAP direct transforming growth factor β-induced tumorigenic phenotypes in breast cancer cells.

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962-970

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10.1016/J.JHEP.2015.05.031

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Richard T Premont

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2015-06-10T00:00:00Z

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278160064

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