Innate immunity defines the capacity of antiviral T cells to limit persistent infection - Wikidata - thesis-toulmin - TriplyDB

Innate immunity defines the capacity of antiviral T cells to limit persistent infection

Innate immunity defines the capacity of antiviral T cells to limit persistent infection

http://www.wikidata.org/entity/Q33902354

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Recognition and Regulation of T Cells by NK Cells "Natural Regulators": NK Cells as Modulators of T Cell Immunity NK Cells in HIV Disease NK Cell and CD4+FoxP3+ Regulatory T Cell Based Therapies for Hematopoietic Stem Cell Engraftment Evasion from NK cell-mediated immune responses by HIV-1 Genomic Modifiers of Natural Killer Cells, Immune Responsiveness and Lymphoid Tissue Remodeling Together Increase Host Resistance to Viral Infection Natural Killer Cell Sensing of Infected Cells Compensates for MyD88 Deficiency but Not IFN-I Activity in Resistance to Mouse Cytomegalovirus Targeting of a natural killer cell receptor family by a viral immunoevasin Boosting vaccine efficacy the natural (killer) way Innate or adaptive immunity? The example of natural killer cells Acute Virus Control Mediated by Licensed NK Cells Sets Primary CD8+ T Cell Dependence on CD27 Costimulation.Self MHC class I-licensed NK cells enhance adaptive CD8 T-cell viral immunity.Licensing delineates helper and effector NK cell subsets during viral infection.Effective control of viral infections by the adaptive immune system requires assistance from innate immunity.Expression pattern of CD11c on lung immune cells after disseminated murine cytomegalovirus infection.Deciphering the role of DC subsets in MCMV infection to better understand immune protection against viral infections Absence of cross-presenting cells in the salivary gland and viral immune evasion confine cytomegalovirus immune control to effector CD4 T cells.Tuning of natural killer cell reactivity by NKp46 and Helios calibrates T cell responses.Murine CD4+ T cell responses are inhibited by cytotoxic T cell-mediated killing of dendritic cells and are restored by antigen transfer.UNC93B1 mediates innate inflammation and antiviral defense in the liver during acute murine cytomegalovirus infection.Dendritic cell editing by activated natural killer cells results in a more protective cancer-specific immune response NK cells and virus-related cancers.Recombinant mouse cytomegalovirus expressing a ligand for the NKG2D receptor is attenuated and has improved vaccine properties.Rapid and sequential quantitation of salivary gland-associated mouse cytomegalovirus in oral lavage IL-10 suppression of NK/DC crosstalk leads to poor priming of MCMV-specific CD4 T cells and prolonged MCMV persistence.Natural killer cell dependent within-host competition arises during multiple MCMV infection: consequences for viral transmission and evolution.NK cells and their ability to modulate T cells during virus infections Interactions of human myeloid cells with natural killer cell subsets in vitro and in vivo Mutiny or scrutiny: NK cell modulation of DC function in HIV-1 infection.Distinct MHC class I-dependent NK cell-activating receptors control cytomegalovirus infection in different mouse strains.A Functional Type I Interferon Pathway Drives Resistance to Cornea Herpes Simplex Virus Type 1 Infection by Recruitment of Leukocytes Cytomegalovirus reactivation in critically ill immunocompetent hosts: a decade of progress and remaining challenges.Constitutive but not inducible attenuation of transforming growth factor β signaling increases natural killer cell responses without directly affecting dendritic cells early after persistent viral infection.Perforin is a critical physiologic regulator of T-cell activation.Functions of liver natural killer cells are dependent on the severity of liver inflammation and fibrosis in chronic hepatitis C.NK cells: immune cross-talk and therapeutic implications.Sequence variations in HIV-1 p24 Gag-derived epitopes can alter binding of KIR2DL2 to HLA-C*03:04 and modulate primary natural killer cell function Yet another role for natural killer cells: cytotoxicity in immune regulation and viral persistence.High IFN-gamma and TNF production by peripheral NK cells of Colombian patients with different clinical presentation of Plasmodium falciparum.The NK cell response to mouse cytomegalovirus infection affects the level and kinetics of the early CD8(+) T-cell response.

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Innate immunity defines the capacity of antiviral T cells to limit persistent infection

http://www.wikidata.org/entity/Q33902354

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Innate immunity defines the capacity of antiviral T cells to limit persistent infection

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Innate immunity defines the capacity of antiviral T cells to limit persistent infection

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Innate immunity defines the capacity of antiviral T cells to limit persistent infection

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Innate immunity defines the capacity of antiviral T cells to limit persistent infection

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Innate immunity defines the capacity of antiviral T cells to limit persistent infection

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Innate immunity defines the capacity of antiviral T cells to limit persistent infection

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Journal of Experimental Medicine

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Innate immunity defines the capacity of antiviral T cells to limit persistent infection

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P2093

Andrea Khong

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Anthony A Scalzo

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Hyacinth Tabarias

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Mariapia A Degli-Esposti

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Matthew E Wikstrom

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Peter Fleming

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Robbert G van der Most

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Valentina Voigt

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P2860

Natural killer cells promote early CD8 T cell responses against cytomegalovirus

Vigorous HIV-1-specific CD4+ T cell responses associated with control of viremia

The effect of the Cmv-1 resistance gene, which is linked to the natural killer cell gene complex, is mediated by natural killer cells

Increased susceptibility to cytomegalovirus infection in beige mutant mice

In vivo depletion of CD11c+ dendritic cells abrogates priming of CD8+ T cells by exogenous cell-associated antigens

Selective down-regulation of the NKG2D ligand H60 by mouse cytomegalovirus m155 glycoprotein

Gamma interferon-dependent clearance of cytomegalovirus infection in salivary glands

Innate partnership of HLA-B and KIR3DL1 subtypes against HIV-1.

Recognition of a virus-encoded ligand by a natural killer cell activation receptor.

Host defense, viruses and apoptosis.

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1333-1343

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scholarly article

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10.1084/JEM.20091193

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6

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207

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Geoffrey R Hill

Christopher E Andoniou

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2010-05-31T00:00:00Z

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44640855

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20513749

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2882831

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