p53 and ATF4 mediate distinct and additive pathways to skeletal muscle atrophy during limb immobilization.
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Role of Inflammation in Muscle Homeostasis and MyogenesisUse of mRNA expression signatures to discover small molecule inhibitors of skeletal muscle atrophy.Skeletal muscle atrophy and the E3 ubiquitin ligases MuRF1 and MAFbx/atrogin-1Post-exercise cold water immersion attenuates acute anabolic signalling and long-term adaptations in muscle to strength training.Identification and Small Molecule Inhibition of an Activating Transcription Factor 4 (ATF4)-dependent Pathway to Age-related Skeletal Muscle Weakness and Atrophy.Spermine oxidase maintains basal skeletal muscle gene expression and fiber size and is strongly repressed by conditions that cause skeletal muscle atrophy.Patients experiencing statin-induced myalgia exhibit a unique program of skeletal muscle gene expression following statin re-challengeControl of skeletal muscle atrophy in response to disuse: clinical/preclinical contentions and fallacies of evidenceAmino Acid Sensing in Skeletal Muscle.Induction of autophagy through the activating transcription factor 4 (ATF4)-dependent amino acid response pathway in maternal skeletal muscle may function as the molecular memory in response to gestational protein restriction to alert offspring to mDifferential expression of perilipin 2 and 5 in human skeletal muscle during aging and their association with atrophy-related genes.Glycine restores the anabolic response to leucine in a mouse model of acute inflammation.Skeletal Muscle Fibre-Specific Knockout of p53 Does Not Reduce Mitochondrial Content or Enzyme Activity.A systematic review of p53 regulation of oxidative stress in skeletal muscle.Prevention of chemotherapy-induced cachexia by ACVR2B ligand blocking has different effects on heart and skeletal muscle.miR-422a suppresses SMAD4 protein expression and promotes resistance to muscle loss.Palmitate-induced ER stress and inhibition of protein synthesis in cultured myotubes does not require Toll-like receptor 4.OPA1 deficiency promotes secretion of FGF21 from muscle that prevents obesity and insulin resistance.Recent advances in mitochondrial turnover during chronic muscle disuse.Natural disease history of the dy2J mouse model of laminin α2 (merosin)-deficient congenital muscular dystrophy.Muscle Atrophy Induced by Mechanical Unloading: Mechanisms and Potential Countermeasures.The Role of p53 in Determining Mitochondrial Adaptations to Endurance Training in Skeletal Muscle
P2860
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P2860
p53 and ATF4 mediate distinct and additive pathways to skeletal muscle atrophy during limb immobilization.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
p53 and ATF4 mediate distinct ...... hy during limb immobilization.
@ast
p53 and ATF4 mediate distinct ...... hy during limb immobilization.
@en
p53 and ATF4 mediate distinct ...... hy during limb immobilization.
@nl
type
label
p53 and ATF4 mediate distinct ...... hy during limb immobilization.
@ast
p53 and ATF4 mediate distinct ...... hy during limb immobilization.
@en
p53 and ATF4 mediate distinct ...... hy during limb immobilization.
@nl
prefLabel
p53 and ATF4 mediate distinct ...... hy during limb immobilization.
@ast
p53 and ATF4 mediate distinct ...... hy during limb immobilization.
@en
p53 and ATF4 mediate distinct ...... hy during limb immobilization.
@nl
P2093
P2860
P1476
p53 and ATF4 mediate distinct ...... hy during limb immobilization.
@en
P2093
Daniel K Fox
Jason M Dierdorff
Kale S Bongers
Michael C Dyle
Scott M Ebert
Steven A Bullard
Steven D Kunkel
P2860
P304
P356
10.1152/AJPENDO.00010.2014
P577
2014-06-03T00:00:00Z