Inflammasomes as mediators of immunity against influenza virus. - Wikidata - thesis-toulmin - TriplyDB

Inflammasomes as mediators of immunity against influenza virus.

Inflammasomes as mediators of immunity against influenza virus.

http://www.wikidata.org/entity/Q34474244

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Activation of P2X(7) receptor by ATP plays an important role in regulating inflammatory responses during acute viral infection Induction of innate immunity and its perturbation by influenza viruses Regulating the adaptive immune response to respiratory virus infection Activation of the NLRP3 inflammasome by IAV virulence protein PB1-F2 contributes to severe pathophysiology and disease Asparagine endopeptidase controls anti-influenza virus immune responses through TLR7 activation Inﬂuenza and Memory T Cells: How to Awake the Force Immune Responses to Influenza Virus and Its Correlation to Age and Inherited Factors Intrinsic antiviral immunity The effector T cell response to influenza infection Viral infection of the lung: host response and sequelae Mannose-binding lectin contributes to deleterious inflammatory response in pandemic H1N1 and avian H9N2 infection.Immunogenetic factors associated with severe respiratory illness caused by zoonotic H1N1 and H5N1 influenza viruses.TLR7 recognition is dispensable for influenza virus A infection but important for the induction of hemagglutinin-specific antibodies in response to the 2009 pandemic split vaccine in mice.A network integration approach to predict conserved regulators related to pathogenicity of influenza and SARS-CoV respiratory viruses.Innate immune response to a H3N2 subtype swine influenza virus in newborn porcine trachea cells, alveolar macrophages, and precision-cut lung slices.Pathogenic potential of interferon αβ in acute influenza infection AIM2 Inflammasome Is Critical for Influenza-Induced Lung Injury and Mortality Geldanamycin Reduces Acute Respiratory Distress Syndrome and Promotes the Survival of Mice Infected with the Highly Virulent H5N1 Influenza Virus.Role of aging on innate responses to viral infections.Conserved host response to highly pathogenic avian influenza virus infection in human cell culture, mouse and macaque model systems.Innate immune recognition of respiratory syncytial virus infection.The Split Virus Influenza Vaccine rapidly activates immune cells through Fcγ receptors.Activation and measurement of NLRP3 inflammasome activity using IL-1β in human monocyte-derived dendritic cells In silico analysis of autoimmune diseases and genetic relationships to vaccination against infectious diseases.Tissue factor, blood coagulation, and beyond: an overview.A novel systems pharmacology model for herbal medicine injection: a case using Reduning injection.Virus-induced NETs--critical component of host defense or pathogenic mediator?Caspase-1 independent viral clearance and adaptive immunity against mucosal respiratory syncytial virus infection.Interleukin-33 upregulation in peripheral leukocytes and CNS of multiple sclerosis patients.Detection of prokaryotic mRNA signifies microbial viability and promotes immunity.IL-1R signaling in dendritic cells replaces pattern-recognition receptors in promoting CD8⁺ T cell responses to influenza A virus Effect of a synbiotic on the response to seasonal influenza vaccination is strongly influenced by degree of immunosenescence Control of antiviral immunity by pattern recognition and the microbiome.Predicting Viral Infection From High-Dimensional Biomarker Trajectories.Induction of memory cytotoxic T cells to influenza A virus and subsequent viral clearance is not modulated by PB1-F2-dependent inflammasome activation.Traditional and new influenza vaccines Towards multiscale modeling of influenza infection The Nlrp3 inflammasome promotes age-related thymic demise and immunosenescence Pattern recognition receptors: sentinels in innate immunity and targets of new vaccine adjuvants.Apoptosis signaling in influenza virus propagation, innate host defense, and lung injury.

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Inflammasomes as mediators of immunity against influenza virus.

http://www.wikidata.org/entity/Q34474244

description

2010 nî lūn-bûn

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2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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name

Inflammasomes as mediators of immunity against influenza virus.

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Inflammasomes as mediators of immunity against influenza virus.

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Inflammasomes as mediators of immunity against influenza virus.

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type

label

Inflammasomes as mediators of immunity against influenza virus.

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Inflammasomes as mediators of immunity against influenza virus.

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Inflammasomes as mediators of immunity against influenza virus.

@nl

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Inflammasomes as mediators of immunity against influenza virus.

@ast

Inflammasomes as mediators of immunity against influenza virus.

@en

Inflammasomes as mediators of immunity against influenza virus.

@nl

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J.IT.2010.11.004

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Trends in Immunology

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Inflammasomes as mediators of immunity against influenza virus.

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Akiko Iwasaki

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Iris K Pang

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P2860

PYPAF7, a novel PYRIN-containing Apaf1-like protein that regulates activation of NF-kappa B and caspase-1-dependent cytokine processing

The inflammasomes

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

NLRs join TLRs as innate sensors of pathogens

AIM2 recognizes cytosolic dsDNA and forms a caspase-1-activating inflammasome with ASC

AIM2 activates the inflammasome and cell death in response to cytoplasmic DNA

An orthogonal proteomic-genomic screen identifies AIM2 as a cytoplasmic DNA sensor for the inflammasome

Interleukin-1 is responsible for acute lung immunopathology but increases survival of respiratory influenza virus infection

Recognition of single-stranded RNA viruses by Toll-like receptor 7

IL-1 acts directly on CD4 T cells to enhance their antigen-driven expansion and differentiation

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34-41

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10.1016/J.IT.2010.11.004

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