Genotoxic stress and cellular stress alter the subcellular distribution of human T-cell leukemia virus type 1 tax through a CRM1-dependent mechanism
about
The HTLV-1 Tax interactomeThe HTLV-1 Tax protein inhibits formation of stress granules by interacting with histone deacetylase 6Tax-1 and Tax-2 similarities and differences: focus on post-translational modifications and NF-κB activation.An RNA interference screen identifies the Deubiquitinase STAMBPL1 as a critical regulator of human T-cell leukemia virus type 1 tax nuclear export and NF-κB activation.Localization and sub-cellular shuttling of HTLV-1 tax with the miRNA machinery.Viral oncogenes, noncoding RNAs, and RNA splicing in human tumor virusesThe four and a half LIM family members are novel interactants of the human T-cell leukemia virus type 1 Tax oncoproteinRole of post-translational modifications of HTLV-1 Tax in NF-κB activation.Move or die: the fate of the Tax oncoprotein of HTLV-1.Ubiquitination of HTLV-I Tax in response to DNA damage regulates nuclear complex formation and nuclear export.Human T-cell leukemia virus type 1 tax attenuates the ATM-mediated cellular DNA damage response.Regulation of the nucleocytoplasmic trafficking of viral and cellular proteins by ubiquitin and small ubiquitin-related modifiers.Human T-cell leukemia virus type 1 Tax relieves repression of proliferating cell nuclear antigen gene expressionAnti-apoptotic effect of Tax: an NF-κB path or a CREB way?Animal models on HTLV-1 and related viruses: what did we learn?Regulation of HTLV-1 tax stability, cellular trafficking and NF-κB activation by the ubiquitin-proteasome pathway.Heat Shock Enhances the Expression of the Human T Cell Leukemia Virus Type-I (HTLV-I) Trans-Activator (Tax) Antigen in Human HTLV-I Infected Primary and Cultured T Cells.The Major Histocompatibility Complex Class II Transactivator CIITA Inhibits the Persistent Activation of NF-κB by the Human T Cell Lymphotropic Virus Type 1 Tax-1 Oncoprotein.The nuclear import of the human T lymphotropic virus type I (HTLV-1) tax protein is carrier- and energy-independent.
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P2860
Genotoxic stress and cellular stress alter the subcellular distribution of human T-cell leukemia virus type 1 tax through a CRM1-dependent mechanism
description
2006 nî lūn-bûn
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2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2006年の論文
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2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Genotoxic stress and cellular ...... ugh a CRM1-dependent mechanism
@ast
Genotoxic stress and cellular ...... ugh a CRM1-dependent mechanism
@en
Genotoxic stress and cellular ...... ugh a CRM1-dependent mechanism
@nl
type
label
Genotoxic stress and cellular ...... ugh a CRM1-dependent mechanism
@ast
Genotoxic stress and cellular ...... ugh a CRM1-dependent mechanism
@en
Genotoxic stress and cellular ...... ugh a CRM1-dependent mechanism
@nl
prefLabel
Genotoxic stress and cellular ...... ugh a CRM1-dependent mechanism
@ast
Genotoxic stress and cellular ...... ugh a CRM1-dependent mechanism
@en
Genotoxic stress and cellular ...... ugh a CRM1-dependent mechanism
@nl
P2860
P356
P1433
P1476
Genotoxic stress and cellular ...... ugh a CRM1-dependent mechanism
@en
P2093
Michael L Gatza
Susan J Marriott
P2860
P304
P356
10.1128/JVI.02270-05
P407
P577
2006-07-01T00:00:00Z