Vav1 dephosphorylation by the tyrosine phosphatase SHP-1 as a mechanism for inhibition of cellular cytotoxicity. - Wikidata - thesis-toulmin - TriplyDB

Vav1 dephosphorylation by the tyrosine phosphatase SHP-1 as a mechanism for inhibition of cellular cytotoxicity.

Vav1 dephosphorylation by the tyrosine phosphatase SHP-1 as a mechanism for inhibition of cellular cytotoxicity.

http://www.wikidata.org/entity/Q35661770

about

An essential function for beta-arrestin 2 in the inhibitory signaling of natural killer cells RasGRP1 is required for human NK cell function Comparative genomics of natural killer cell receptor gene clusters.Newtonian cell interactions shape natural killer cell education Phosphorylation-mediated regulation of GEFs for RhoA Cell biological steps and checkpoints in accessing NK cell cytotoxicity Current perspectives on natural killer cell education and tolerance: emerging roles for inhibitory receptors Inhibitory receptor signaling via tyrosine phosphorylation of the adaptor Crk Up on the tightrope: natural killer cell activation and inhibition Association of 3BP2 with SHP-1 regulates SHP-1-mediated production of TNF-α in RBL-2H3 cells.Rapid activation receptor- or IL-2-induced lytic granule convergence in human natural killer cells requires Src, but not downstream signaling Toll-like receptor ligands sensitize B-cell receptor signalling by reducing actin-dependent spatial confinement of the receptor.Identification of CLEC12B, an inhibitory receptor on myeloid cells.Recruitment of activation receptors at inhibitory NK cell immune synapses Ly49C-dependent control of MCMV Infection by NK cells is cis-regulated by MHC Class I molecules Synergistic signals for natural cytotoxicity are required to overcome inhibition by c-Cbl ubiquitin ligase Expression of CD226 is associated to but not required for NK cell education.STAT3- and DNA methyltransferase 1-mediated epigenetic silencing of SHP-1 tyrosine phosphatase tumor suppressor gene in malignant T lymphocytes Substrate specificity of protein tyrosine phosphatases 1B, RPTPα, SHP-1, and SHP-2.Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis Cytotoxic immunological synapses.Functional requirements for inhibitory signal transmission by the immunomodulatory receptor CD300a.SHP-1 in T cells limits the production of CD8 effector cells without impacting the formation of long-lived central memory cells Siglec-5 (CD170) can mediate inhibitory signaling in the absence of immunoreceptor tyrosine-based inhibitory motif phosphorylation.Balancing natural killer cell activation through paired receptors.Inhibitory signaling blocks activating receptor clustering and induces cytoskeletal retraction in natural killer cells A single amino acid change in inhibitory killer cell Ig-like receptor results in constitutive receptor self-association and phosphorylation.Transcriptional profiling of human monocytes identifies the inhibitory receptor CD300a as regulator of transendothelial migration.Natural killer cell responses to viral infection.NK cells: immune cross-talk and therapeutic implications.Vav1 Regulates T-Cell Activation through a Feedback Mechanism and Crosstalk between the T-Cell Receptor and CD28.Activation of natural killer cells: underlying molecular mechanisms revealed.Uncoupling of T-cell effector functions by inhibitory killer immunoglobulin-like receptors.The adaptor protein Crk controls activation and inhibition of natural killer cells.The Size of Activating and Inhibitory Killer Ig-like Receptor Nanoclusters Is Controlled by the Transmembrane Sequence and Affects Signaling Positive and negative signaling through SLAM receptors regulate synapse organization and thresholds of cytolysis.Formation of a WIP-, WASp-, actin-, and myosin IIA-containing multiprotein complex in activated NK cells and its alteration by KIR inhibitory signaling.Microclusters of inhibitory killer immunoglobulin-like receptor signaling at natural killer cell immunological synapses Signaling pathways in aged T cells - a reflection of T cell differentiation, cell senescence and host environment.NK Cell Proliferation Induced by IL-15 Transpresentation Is Negatively Regulated by Inhibitory Receptors.

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Vav1 dephosphorylation by the tyrosine phosphatase SHP-1 as a mechanism for inhibition of cellular cytotoxicity.

http://www.wikidata.org/entity/Q35661770

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MCB.23.17.6291-6299.2003

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Molecular and Cellular Biology

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Carsten Watzl

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Christopher C Stebbins

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Daniel D Billadeau

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Deborah N Burshtyn

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Eric O Long

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P2860

Activation of NK cells and T cells by NKG2D, a receptor for stress-inducible MICA

Pivotal role of phosphoinositide-3 kinase in regulation of cytotoxicity in natural killer cells

Cutting edge: differential segregation of the SRC homology 2-containing protein tyrosine phosphatase-1 within the early NK cell immune synapse distinguishes noncytolytic from cytolytic interactions

Development of "substrate-trapping" mutants to identify physiological substrates of protein tyrosine phosphatases

Identification of major binding proteins and substrates for the SH2-containing protein tyrosine phosphatase SHP-1 in macrophages

Regulatory and signaling properties of the Vav family

The human natural killer cell immune synapse

DAP10 and DAP12 form distinct, but functionally cooperative, receptor complexes in natural killer cells

Structural basis for substrate specificity of protein-tyrosine phosphatase SHP-1

Structural basis for relief of autoinhibition of the Dbl homology domain of proto-oncogene Vav by tyrosine phosphorylation

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