Peripheral innate immune challenge exaggerated microglia activation, increased the number of inflammatory CNS macrophages, and prolonged social withdrawal in socially defeated mice.
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Stress and neuroinflammation: a systematic review of the effects of stress on microglia and the implications for mental illnessNeuroimmune mechanisms of stress: sex differences, developmental plasticity, and implications for pharmacotherapy of stress-related diseaseSurveillance, phagocytosis, and inflammation: how never-resting microglia influence adult hippocampal neurogenesisAcute and chronic stress-induced disturbances of microglial plasticity, phenotype and functionNeuroinflammation at the interface of depression and cardiovascular disease: Evidence from rodent models of social stressStress-induced neuroinflammatory priming: A liability factor in the etiology of psychiatric disordersChronic exposure to exogenous glucocorticoids primes microglia to pro-inflammatory stimuli and induces NLRP3 mRNA in the hippocampusFound in translation: Understanding the biology and behavior of experimental traumatic brain injuryStress-induced neuroinflammatory priming is time of day dependent.Re-establishment of anxiety in stress-sensitized mice is caused by monocyte trafficking from the spleen to the brain.Neuroimmune Interactions in Schizophrenia: Focus on Vagus Nerve Stimulation and Activation of the Alpha-7 Nicotinic Acetylcholine ReceptorTGFβ produced by IL-10 redirected astrocytes attenuates microglial activationIL-4 signaling drives a unique arginase+/IL-1β+ microglia phenotype and recruits macrophages to the inflammatory CNS: consequences of age-related deficits in IL-4Rα after traumatic spinal cord injury.Dietary intervention rescues maternal obesity induced behavior deficits and neuroinflammation in offspringMicroglial dysregulation in psychiatric diseaseStress induces the danger-associated molecular pattern HMGB-1 in the hippocampus of male Sprague Dawley rats: a priming stimulus of microglia and the NLRP3 inflammasome.Methylene blue attenuates traumatic brain injury-associated neuroinflammation and acute depressive-like behavior in mice.Monocyte trafficking to the brain with stress and inflammation: a novel axis of immune-to-brain communication that influences mood and behaviorSocial defeat promotes a reactive endothelium in a brain region-dependent manner with increased expression of key adhesion molecules, selectins and chemokines associated with the recruitment of myeloid cells to the brainPrebiotic administration normalizes lipopolysaccharide (LPS)-induced anxiety and cortical 5-HT2A receptor and IL1-β levels in male micePeripheral and central effects of repeated social defeat stress: monocyte trafficking, microglial activation, and anxiety.Sequential activation of microglia and astrocyte cytokine expression precedes increased Iba-1 or GFAP immunoreactivity following systemic immune challenge.Stress-induced neuroinflammation is mediated by GSK3-dependent TLR4 signaling that promotes susceptibility to depression-like behavior.LPS-induced Murine Neuroinflammation Model: Main Features and Suitability for Pre-clinical Assessment of NutraceuticalsCognitive deficits develop 1month after diffuse brain injury and are exaggerated by microglia-associated reactivity to peripheral immune challenge.Crosstalk between endocannabinoid and immune systems: a potential dysregulation in depression?Sex differences in stress-induced social withdrawal: independence from adult gonadal hormones and inhibition of female phenotype by corncob beddingStress-induced recruitment of bone marrow-derived monocytes to the brain promotes anxiety-like behavior.Programmed symptoms: disparate effects united by purpose.Neonatal E. coli infection causes neuro-behavioral deficits associated with hypomyelination and neuronal sequestration of ironSocial defeat induces depressive-like states and microglial activation without involvement of peripheral macrophagesVirus Infections on Prion Diseased Mice Exacerbate Inflammatory Microglial Response.Knockdown of interleukin-1 receptor type-1 on endothelial cells attenuated stress-induced neuroinflammation and prevented anxiety-like behavior.Sociality and sickness: have cytokines evolved to serve social functions beyond times of pathogen exposure?Neuroinflammatory and cognitive consequences of combined radiation and immunotherapy in a novel preclinical model.Design, synthesis and evaluation in an LPS rodent model of neuroinflammation of a novel 18F-labelled PET tracer targeting P2X7.Animal inflammation-based models of depression and their application to drug discovery.Neuroinflammation: the devil is in the details.Integrating neuroimmune systems in the neurobiology of depression.Repeated Social Defeat, Neuroinflammation, and Behavior: Monocytes Carry the Signal.
P2860
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P2860
Peripheral innate immune challenge exaggerated microglia activation, increased the number of inflammatory CNS macrophages, and prolonged social withdrawal in socially defeated mice.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Peripheral innate immune chall ...... wal in socially defeated mice.
@ast
Peripheral innate immune chall ...... wal in socially defeated mice.
@en
type
label
Peripheral innate immune chall ...... wal in socially defeated mice.
@ast
Peripheral innate immune chall ...... wal in socially defeated mice.
@en
prefLabel
Peripheral innate immune chall ...... wal in socially defeated mice.
@ast
Peripheral innate immune chall ...... wal in socially defeated mice.
@en
P2093
P2860
P1476
Peripheral innate immune chall ...... awal in socially defeated mice
@en
P2093
Ann M Pacenta
Ashley M Fenn
Eric S Wohleb
Jonathan P Godbout
Nicole D Powell
P2860
P304
P356
10.1016/J.PSYNEUEN.2012.02.003
P577
2012-03-02T00:00:00Z