Absence of amyloid β oligomers at the postsynapse and regulated synaptic Zn2+ in cognitively intact aged individuals with Alzheimer's disease neuropathology.
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The Role of Amyloid-β Oligomers in Toxicity, Propagation, and ImmunotherapyTau Oligomers: The Toxic Player at Synapses in Alzheimer's DiseaseAlzheimer's disease and insulin resistance: translating basic science into clinical applicationsSynaptopathies: synaptic dysfunction in neurological disorders - A review from students to studentsComputational identification of potential multitarget treatments for ameliorating the adverse effects of amyloid-β on synaptic plasticitySpatial memory deficits in a mouse model of late-onset Alzheimer's disease are caused by zinc supplementation and correlate with amyloid-beta levelsSoluble amyloid-β oligomers as synaptotoxins leading to cognitive impairment in Alzheimer's diseasePeripheral adipose tissue insulin resistance alters lipid composition and function of hippocampal synapsesPreserved neurogenesis in non-demented individuals with AD neuropathology.Synaptic Amyloid-β Oligomers Precede p-Tau and Differentiate High Pathology Control Cases.Reduced Incidence of Dementia in Solid Organ Transplant Patients Treated with Calcineurin InhibitorsAnalysis of Zinc-Exporters Expression in Prostate Cancer.Soluble Amyloid-beta Aggregates from Human Alzheimer's Disease Brains.Zinc and the aging brainBiological metals and metal-targeting compounds in major neurodegenerative diseases.The Physiological, Biochemical, and Molecular Roles of Zinc Transporters in Zinc Homeostasis and Metabolism.Relationship between Zinc (Zn (2+) ) and Glutamate Receptors in the Processes Underlying Neurodegeneration.Linking insulin with Alzheimer's disease: emergence as type III diabetes.Non-Demented Individuals with Alzheimer's Disease Neuropathology: Resistance to Cognitive Decline May Reveal New Treatment Strategies.Cerebral hypoperfusion and glucose hypometabolism: Key pathophysiological modulators promote neurodegeneration, cognitive impairment, and Alzheimer's disease.Cytokines and Cytokine Receptors Involved in the Pathogenesis of Alzheimer's Disease.Near infrared light decreases synaptic vulnerability to amyloid beta oligomers.Endothelial LRP1 - A Potential Target for the Treatment of Alzheimer's Disease : Theme: Drug Discovery, Development and Delivery in Alzheimer's Disease Guest Editor: Davide Brambilla.Non-targeted lipidomics of CSF and frontal cortex grey and white matter in control, mild cognitive impairment, and Alzheimer's disease subjects.Challenges for Alzheimer's Disease Therapy: Insights from Novel Mechanisms Beyond Memory Defects.Postsynaptic Proteome of Non-Demented Individuals with Alzheimer's Disease Neuropathology
P2860
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P2860
Absence of amyloid β oligomers at the postsynapse and regulated synaptic Zn2+ in cognitively intact aged individuals with Alzheimer's disease neuropathology.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Absence of amyloid β oligomers ...... imer's disease neuropathology.
@ast
Absence of amyloid β oligomers ...... imer's disease neuropathology.
@en
type
label
Absence of amyloid β oligomers ...... imer's disease neuropathology.
@ast
Absence of amyloid β oligomers ...... imer's disease neuropathology.
@en
prefLabel
Absence of amyloid β oligomers ...... imer's disease neuropathology.
@ast
Absence of amyloid β oligomers ...... imer's disease neuropathology.
@en
P2093
P2860
P356
P1476
Absence of amyloid β oligomers ...... imer's disease neuropathology.
@en
P2093
Giulio Taglialatela
Lindsay C Reese
Nicole L Bjorklund
V-M Sadagoparamanujam
Valeria Ghirardi
P2860
P2888
P356
10.1186/1750-1326-7-23
P577
2012-05-28T00:00:00Z
P5875
P6179
1047619642