The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury. - Wikidata - thesis-toulmin - TriplyDB

The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.

The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.

http://www.wikidata.org/entity/Q36472435

about

Mitochondrial VDAC1: A Key Gatekeeper as Potential Therapeutic Target Inositol 1,4,5-trisphosphate receptors and their protein partners as signalling hubs.Sarcoplasmic reticulum-mitochondria communication in cardiovascular pathophysiology.Potential Roles of Mitochondria-Associated ER Membranes (MAMs) in Traumatic Brain Injury.Role of glycogen synthase kinase following myocardial infarction and ischemia-reperfusion.Protein kinase D activation induces mitochondrial fragmentation and dysfunction in cardiomyocytes.Alterations in Ca2+ Signalling via ER-Mitochondria Contact Site Remodelling in Cancer.Reciprocal Interactions of Mitochondria and the Neuroimmunoendocrine System in Neurodegenerative Disorders: An Important Role for Melatonin Regulation.Melatonin-Induced Protective Effects on Cardiomyocytes Against Reperfusion Injury Partly Through Modulation of IP3R and SERCA2a Via Activation of ERK1.The role of endoplasmic reticulum-mitochondria contact sites in the control of glucose homeostasis: an update.ER–Mitochondria Microdomains in Cardiac Ischemia–Reperfusion Injury: A Fresh Perspective.Consensus report of the 8 and 9th Weinman Symposia on Gene x Environment Interaction in carcinogenesis: novel opportunities for precision medicine

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P2860

The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.

http://www.wikidata.org/entity/Q36472435

description

2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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2015年學術文章

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2015年學術文章

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name

The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.

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The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.

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type

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The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.

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The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.

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prefLabel

The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.

@ast

The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.

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The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury.

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A Durand

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C Crola Da Silva

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F Van Coppenolle

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S-S Sheu

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P2860

Ca(2+) transfer from the ER to mitochondria: when, how and why

Calcium-mediated cell death during myocardial reperfusion

Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase B

Cardiac excitation-contraction coupling

Myocardial reperfusion: a double-edged sword?

Sigma-1 receptor chaperones at the ER-mitochondrion interface regulate Ca(2+) signaling and cell survival

Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo

Molecular cloning and expression of glycogen synthase kinase-3/factor A

Methods in cardiomyocyte isolation, culture, and gene transfer.

Translocation of glycogen synthase kinase-3β (GSK-3β), a trigger of permeability transition, is kinase activity-dependent and mediated by interaction with voltage-dependent anion channel 2 (VDAC2).

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313-322

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scholarly article

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10.1038/CDD.2015.101

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2

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23

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Mélanie Paillard

Muhammad Rizwan Alam

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2015-07-24T00:00:00Z

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280317453

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1005888366

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reperfusion injury

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