Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma. - Wikidata - thesis-toulmin - TriplyDB

Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma.

Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma.

http://www.wikidata.org/entity/Q37609446

about

Evasion of anti-growth signaling: A key step in tumorigenesis and potential target for treatment and prophylaxis by natural compounds.Cell density sensing alters TGF-β signaling in a cell-type-specific manner, independent from Hippo pathway activation Genetic variants in Hippo pathway genes YAP1, TEAD1 and TEAD4 are associated with melanoma-specific survival Nicotine activates YAP1 through nAChRs mediated signaling in esophageal squamous cell cancer (ESCC).FGF8 promotes colorectal cancer growth and metastasis by activating YAP1.Decoding the regulatory landscape of melanoma reveals TEADS as regulators of the invasive cell state.Cell growth density modulates cancer cell vascular invasion via Hippo pathway activity and CXCR2 signaling.Actin remodeling confers BRAF inhibitor resistance to melanoma cells through YAP/TAZ activation.Yap1 promotes the survival and self-renewal of breast tumor initiating cells via inhibiting Smad3 signaling Cell density regulates cancer metastasis via the Hippo pathway.CYR61 (CCN1) is a metastatic biomarker of gastric cardia adenocarcinoma.An updated review of mechanotransduction in skin disorders: transcriptional regulators, ion channels, and microRNAs.Control of Proliferation and Cancer Growth by the Hippo Signaling Pathway.Yes-associated protein (YAP) expression is involved in epithelial-mesenchymal transition in hepatocellular carcinoma.YAP/TAZ at the Roots of Cancer.Role of YAP/TAZ transcriptional regulators in resistance to anti-cancer therapies.Focus on cutaneous and uveal melanoma specificities.Hippo signaling promotes JNK-dependent cell migration.Inhibition of YAP function overcomes BRAF inhibitor resistance in melanoma cancer stem cells.Yes-Associated Protein (YAP) Promotes Tumorigenesis in Melanoma Cells Through Stimulation of Low-Density Lipoprotein Receptor-Related Protein 1 (LRP1).Clinicopathological and prognostic significance of Yes-associated protein expression in hepatocellular carcinoma and hepatic cholangiocarcinoma.Collagen abundance controls melanoma phenotypes through lineage-specific microenvironment sensing.Expression of Hippo pathway genes and their clinical significance in colon adenocarcinoma.YAP/TAZ Activation as a Target for Treating Metastatic Cancer.Fascin induces melanoma tumorigenesis and stemness through regulating the Hippo pathway.

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Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma.

http://www.wikidata.org/entity/Q37609446

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 29 July 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma.

@en

Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma.

@nl

type

label

Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma.

@en

Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma.

@nl

prefLabel

Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma.

@en

Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma.

@nl

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Journal of Investigative Dermatology

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Pro-invasive activity of the Hippo pathway effectors YAP and TAZ in cutaneous melanoma.

@en

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Alain Mauviel

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Cristèle Gilbert

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Flore Nallet-Staub

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Ling Li

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Marius Sudol

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Sophie Dodier

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Véronique Bataille

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Véronique Marsaud

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P2860

Yap1 acts downstream of α-catenin to control epidermal proliferation

TEAD transcription factors mediate the function of TAZ in cell growth and epithelial-mesenchymal transition

TAZ controls Smad nucleocytoplasmic shuttling and regulates human embryonic stem-cell self-renewal

TEAD mediates YAP-dependent gene induction and growth control

Genetic and pharmacological disruption of the TEAD-YAP complex suppresses the oncogenic activity of YAP

The hippo signaling pathway in development and cancer

Structures of YAP protein domains reveal promising targets for development of new cancer drugs

TEAD/TEF transcription factors utilize the activation domain of YAP65, a Src/Yes-associated protein localized in the cytoplasm

Identification, basic characterization and evolutionary analysis of differentially spliced mRNA isoforms of human YAP1 gene

Transforming properties of YAP, a candidate oncogene on the chromosome 11q22 amplicon

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123-132

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scholarly article

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10.1038/JID.2013.319

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1

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134

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Meenhard Herlyn

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2013-07-29T00:00:00Z

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253339328

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3938155

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