about
Producer T cells: Using genetically engineered T cells as vehicles to generate and deliver therapeutics to tumorsSTAT3, a Key Parameter of Cytokine-Driven Tissue Protection during Sterile Inflammation - the Case of Experimental Acetaminophen (Paracetamol)-Induced Liver DamageNovel immunotherapeutic approaches for the treatment of acute leukemia (myeloid and lymphoblastic)The potential of radiotherapy to enhance the efficacy of renal cell carcinoma therapyTargeting the innate immune system as immunotherapy for acute myeloid leukemiaSignal transducer and activator of transcription 3 in myeloid-derived suppressor cells: an opportunity for cancer therapyAdipose-derived mesenchymal stem cells promote osteosarcoma proliferation and metastasis by activating the STAT3 pathway.Myeloid STAT3 promotes formation of colitis-associated colorectal cancer in miceTranscriptional regulation of myeloid-derived suppressor cells.CD45 Phosphatase Inhibits STAT3 Transcription Factor Activity in Myeloid Cells and Promotes Tumor-Associated Macrophage DifferentiationActivated T cells sustain myeloid-derived suppressor cell-mediated immune suppression.Relevance of Interferon Regulatory Factor-8 Expression in Myeloid-Tumor Interactions.Autophagy Differentially Regulates Distinct Breast Cancer Stem-like Cells in Murine Models via EGFR/Stat3 and Tgfβ/Smad Signaling.Gemcitabine reduces MDSCs, tregs and TGFβ-1 while restoring the teff/treg ratio in patients with pancreatic cancerSTAT3: the "Achilles" heel for AML?Epithelial ovarian cancer-secreted exosomal miR-222-3p induces polarization of tumor-associated macrophages.Inflammation and Cancer: Extra- and Intracellular Determinants of Tumor-Associated Macrophages as Tumor Promoters.The Multifaceted Roles of STAT3 Signaling in the Progression of Prostate Cancer.STAT3: An Anti-Invasive Factor in Colorectal Cancer?Restoring Anticancer Immune Response by Targeting Tumor-Derived Exosomes With a HSP70 Peptide Aptamer.Myeloid-derived suppressor cells as effectors of immune suppression in cancer.Nanomedicine Strategies to Target Tumor-Associated Macrophages.The alpha 7 nicotinic receptor agonist PHA-543613 hydrochloride inhibits Porphyromonas gingivalis-induced expression of interleukin-8 by oral keratinocytesDevelopmental Bisphenol A Exposure Modulates Immune-Related Diseases.Identification of inhibitors of myeloid-derived suppressor cells activity through phenotypic chemical screening.Tumor Cell-Independent Estrogen Signaling Drives Disease Progression through Mobilization of Myeloid-Derived Suppressor Cells.STAT3 deficiency prevents hepatocarcinogenesis and promotes biliary proliferation in thioacetamide-induced liver injury.Myeloid-derived suppressor cells coming of age.The role of STAT3 in leading the crosstalk between human cancers and the immune system.Blockade of Host β2-Adrenergic Receptor Enhances Graft-versus-Tumor Effect through Modulating APCs.Tumor-targeted delivery of sunitinib base enhances vaccine therapy for advanced melanoma by remodeling the tumor microenvironment.Down-regulation of STAT3 induces the apoptosis and G1 cell cycle arrest in esophageal carcinoma ECA109 cells.Myeloid p38α signaling promotes intestinal IGF-1 production and inflammation-associated tumorigenesis.Contribution of STAT3 to Inflammatory and Fibrotic Diseases and Prospects for its Targeting for TreatmentMIS416 as a siRNA Delivery System with the Ability to Target Antigen-Presenting Cells
P2860
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P2860
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh
2013年學術文章
@zh-hant
name
STAT3 activation: A key factor in tumor immunoescape.
@en
type
label
STAT3 activation: A key factor in tumor immunoescape.
@en
prefLabel
STAT3 activation: A key factor in tumor immunoescape.
@en
P2860
P356
P1433
P1476
STAT3 activation: A key factor in tumor immunoescape.
@en
P2093
Frédérique Végran
Hélène Berger
P2860
P304
P356
10.4161/JKST.23010
P577
2013-01-01T00:00:00Z