Mutation of ATF4 mediates resistance of neuronal cell lines against oxidative stress by inducing xCT expression.
about
ATF4 protects against neuronal death in cellular Parkinson's disease models by maintaining levels of parkinCommunity structure analysis of transcriptional networks reveals distinct molecular pathways for early- and late-onset temporal lobe epilepsy with childhood febrile seizuresGene expression regulation by upstream open reading frames and human diseasePhosphoinositide 3-kinases upregulate system xc(-) via eukaryotic initiation factor 2α and activating transcription factor 4 - A pathway active in glioblastomas and epilepsyTranscriptional and metabolic adaptation of human neurons to the mitochondrial toxicant MPP(+)Midnolin is a novel regulator of parkin expression and is associated with Parkinson's DiseaseMitochondrial function and energy metabolism in neuronal HT22 cells resistant to oxidative stress.Redox regulation in stem-like cancer cells by CD44 variant isoforms.Main path and byways: non-vesicular glutamate release by system xc(-) as an important modifier of glutamatergic neurotransmission.The effect of Bacopa monnieri on gene expression levels in SH-SY5Y human neuroblastoma cells.Nrf2- and ATF4-dependent upregulation of xCT modulates the sensitivity of T24 bladder carcinoma cells to proteasome inhibition.The role of Ca2+ in cell death caused by oxidative glutamate toxicity and ferroptosis.Cystathionine is a novel substrate of cystine/glutamate transporter: implications for immune function.Methylglyoxal, the foe and friend of glyoxalase and Trx/TrxR systems in HT22 nerve cells.β1-Integrin Deletion From the Lens Activates Cellular Stress Responses Leading to Apoptosis and FibrosisImplication of the glutamate-cystine antiporter xCT in schizophrenia cases linked to impaired GSH synthesis.Inhibition of HIF-prolyl-4-hydroxylases prevents mitochondrial impairment and cell death in a model of neuronal oxytosis.Histone deacetylases inhibition by SAHA/Vorinostat normalizes the glioma microenvironment via xCT equilibration.p53 Suppresses Metabolic Stress-Induced Ferroptosis in Cancer Cells.Guanabenz promotes neuronal survival via enhancement of ATF4 and parkin expression in models of Parkinson disease.Oxytosis/Ferroptosis-(Re-) Emerging Roles for Oxidative Stress-Dependent Non-apoptotic Cell Death in Diseases of the Central Nervous System.
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P2860
Mutation of ATF4 mediates resistance of neuronal cell lines against oxidative stress by inducing xCT expression.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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name
Mutation of ATF4 mediates resi ...... ss by inducing xCT expression.
@en
Mutation of ATF4 mediates resi ...... ss by inducing xCT expression.
@nl
type
label
Mutation of ATF4 mediates resi ...... ss by inducing xCT expression.
@en
Mutation of ATF4 mediates resi ...... ss by inducing xCT expression.
@nl
prefLabel
Mutation of ATF4 mediates resi ...... ss by inducing xCT expression.
@en
Mutation of ATF4 mediates resi ...... ss by inducing xCT expression.
@nl
P2093
P2860
P356
P1476
Mutation of ATF4 mediates resi ...... ess by inducing xCT expression
@en
P2093
P2860
P2888
P304
P356
10.1038/CDD.2011.165
P50
P577
2011-11-18T00:00:00Z