Selenium compounds activate early barriers of tumorigenesis. - Wikidata - thesis-toulmin - TriplyDB

Selenium compounds activate early barriers of tumorigenesis.

Selenium compounds activate early barriers of tumorigenesis.

http://www.wikidata.org/entity/Q39739211

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Current Knowledge on the Importance of Selenium in Food for Living Organisms: A Review Evasion of anti-growth signaling: A key step in tumorigenesis and potential target for treatment and prophylaxis by natural compounds.Selenium cytotoxicity in cancer Dietary phytochemicals, HDAC inhibition, and DNA damage/repair defects in cancer cells Sodium selenide toxicity is mediated by O2-dependent DNA breaks The function and significance of SELENBP1 downregulation in human bronchial epithelial carcinogenic process Methylseleninic acid sensitizes Notch3-activated OVCA429 ovarian cancer cells to carboplatin Nuclear-cytoplasmic trafficking of NTF2, the nuclear import receptor for the RanGTPase, is subjected to regulation.Selenium suppresses leukemia through the action of endogenous eicosanoids.Selenium compounds activate ATM-dependent DNA damage response via the mismatch repair protein hMLH1 in colorectal cancer cells.Neutralization by metal ions of the toxicity of sodium selenide Selenoprotein H suppresses cellular senescence through genome maintenance and redox regulation Nutrition and aging Methylseleninic Acid Superactivates p53-Senescence Cancer Progression Barrier in Prostate Lesions of Pten-Knockout Mouse.Enhancement of NAD⁺-dependent SIRT1 deacetylase activity by methylselenocysteine resets the circadian clock in carcinogen-treated mammary epithelial cells.Cancer chemoprevention research with selenium in the post-SELECT era: Promises and challenges.Selenium supranutrition: are the potential benefits of chemoprevention outweighed by the promotion of diabetes and insulin resistance?Combination of fenretinide and selenite inhibits proliferation and induces apoptosis in ovarian cancer cells.Selenite inhibits glutamine metabolism and induces apoptosis by regulating GLS1 protein degradation via APC/C-CDH1 pathway in colorectal cancer cells.Selenium compounds, apoptosis and other types of cell death: an overview for cancer therapy.Nuclear selenoproteins and genome maintenance.Expression profiling indicating low selenium-sensitive microRNA levels linked to cell cycle and cell stress response pathways in the CaCo-2 cell line.Methylseleninic acid suppresses pancreatic cancer growth involving multiple pathways.Effect of dietary selenium and cancer cell xenograft on peripheral T and B lymphocytes in adult nude mice.Effects of ebselen and N-acetyl cysteine on replicative aging of primary human fibroblast strains.

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Selenium compounds activate early barriers of tumorigenesis.

http://www.wikidata.org/entity/Q39739211

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

@wuu

2010年学术文章

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2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

@yue

2010年學術文章

@zh

2010年學術文章

@zh-hant

name

Selenium compounds activate early barriers of tumorigenesis.

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Selenium compounds activate early barriers of tumorigenesis.

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type

label

Selenium compounds activate early barriers of tumorigenesis.

@en

Selenium compounds activate early barriers of tumorigenesis.

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prefLabel

Selenium compounds activate early barriers of tumorigenesis.

@en

Selenium compounds activate early barriers of tumorigenesis.

@nl

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JBC.M109.088781

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Journal of Biological Chemistry

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Selenium compounds activate early barriers of tumorigenesis.

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Mandy M Kang

http://www.w3.org/2001/XMLSchema#string

Min Wu

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Norberta W Schoene

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Wen-Hsing Cheng

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P2860

Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a)

Role of the p53/p21 system in the response of human colon carcinoma cells to Doxorubicin

Histone H2AX is phosphorylated in an ATR-dependent manner in response to replicational stress

DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation

Regulation of oxidative stress by ATM is required for self-renewal of haematopoietic stem cells

Free radicals, metals and antioxidants in oxidative stress-induced cancer

ATM and related protein kinases: safeguarding genome integrity

Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions

DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis

Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

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12055-12062

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10.1074/JBC.M109.088781

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16

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285

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