ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes.
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Mitochondrial dysfunction in ataxia-telangiectasiaTargeting New Candidate Genes by Small Molecules Approaching Neurodegenerative DiseasesThe identification of zebrafish mutants showing alterations in senescence-associated biomarkersp38 MAPK-Mediated Bmi-1 down-regulation and defective proliferation in ATM-deficient neural stem cells can be restored by Akt activation.The ATM cofactor ATMIN protects against oxidative stress and accumulation of DNA damage in the aging brain.Dengue-induced autophagy, virus replication and protection from cell death require ER stress (PERK) pathway activationA Model for p38MAPK-Induced Astrocyte SenescenceATM kinase inhibition in glial cells activates the innate immune response and causes neurodegeneration in Drosophila.Brain and induced pluripotent stem cell-derived neural stem cells as an in vitro model of neurodegeneration in ataxia-telangiectasia.ATM-deficient human neural stem cells as an in vitro model system to study neurodegeneration.Mutation of ataxia-telangiectasia mutated is associated with dysfunctional glutathione homeostasis in cerebellar astroglia.Neurodegeneration in ataxia-telangiectasia: Multiple roles of ATM kinase in cellular homeostasis.NFBD1/MDC1 regulates Cav1 and Cav2 independently of DNA damage and p53.Neuroprotective effects of the drug GVT (monosodium luminol) are mediated by the stabilization of Nrf2 in astrocytesSelenium compounds activate early barriers of tumorigenesis.ATM is required for SOD2 expression and homeostasis within the mammary gland.Retrovirus-induced oxidative stress with neuroimmunodegeneration is suppressed by antioxidant treatment with a refined monosodium alpha-luminol (Galavit).Targeting p38 mitogen-activated protein kinase signaling restores subventricular zone neural stem cells and corrects neuromotor deficits in Atm knockout mouse.Individual Cytokines Modulate the Neurological Symptoms of ATM Deficiency in a Region Specific Manner(1,2,3)Activation of AMP-activated protein kinase in cerebella of Atm-/- mice is attributable to accumulation of reactive oxygen species.Oxidative stress is linked to ERK1/2-p16 signaling-mediated growth defect in ATM-deficient astrocytes.Functional and molecular defects of hiPSC-derived neurons from patients with ATM deficiencyA role for vascular deficiency in retinal pathology in a mouse model of ataxia-telangiectasia.ATM-dependent pathways of chromatin remodelling and oxidative DNA damage responses.
P2860
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P2860
ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes.
description
2005 nî lūn-bûn
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2005年の論文
@ja
2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年學術文章
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2005年學術文章
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name
ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes.
@en
ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes.
@nl
type
label
ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes.
@en
ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes.
@nl
prefLabel
ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes.
@en
ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes.
@nl
P2093
P2860
P1476
ATM deficiency induces oxidative stress and endoplasmic reticulum stress in astrocytes.
@en
P2093
Mingshan Yan
Paul K Y Wong
Virginia L Scofield
Wenan Qiang
William S Lynn
P2860
P2888
P304
P356
10.1038/LABINVEST.3700354
P577
2005-12-01T00:00:00Z