BBS-induced ciliary defect enhances adipogenesis, causing paradoxical higher-insulin sensitivity, glucose usage, and decreased inflammatory response
about
Pharmacological modulation of the retinal unfolded protein response in Bardet-Biedl syndrome reduces apoptosis and preserves light detection abilityPrimary cilia in pancreatic development and disease.Whole Exome Sequencing Identifies a Novel and a Recurrent Mutation in BBS2 Gene in a Family with Bardet-Biedl SyndromeRegulation of Insulin Receptor Trafficking by Bardet Biedl Syndrome Proteins.Muscle transcriptome response to ACTH administration in a free-ranging marine mammal.The BBSome Controls Energy Homeostasis by Mediating the Transport of the Leptin Receptor to the Plasma Membrane.Comparing the Bbs10 complete knockout phenotype with a specific renal epithelial knockout one highlights the link between renal defects and systemic inactivation in micePrimary cilia in energy balance signaling and metabolic disorder.Depletion of BBS Protein LZTFL1 Affects Growth and Causes Retinal Degeneration in Mice.Acute dietary fat intake initiates alterations in energy metabolism and insulin resistance.Canonical and non-canonical Hedgehog signalling and the control of metabolism.Metabolic regulation and energy homeostasis through the primary Cilium.Bardet-Biedl syndrome: Is it only cilia dysfunction?BBS4 regulates the expression and secretion of FSTL1, a protein that participates in ciliogenesis and the differentiation of 3T3-L1.Obesity-associated sympathetic overactivity in children and adolescents: the role of catecholamine resistance in lipid metabolism.The cilium: a cellular antenna with an influence on obesity risk.Primary Cilia and Coordination of Receptor Tyrosine Kinase (RTK) and Transforming Growth Factor β (TGF-β) Signaling.Cilia and Obesity.Insulin regulates Bbs4 during adipogenesis.New Roles of the Primary Cilium in Autophagy.Adipogenic Differentiation of hMSCs is Mediated by Recruitment of IGF-1r Onto the Primary Cilium Associated With Cilia Elongation.The primary cilium is necessary for the differentiation and the maintenance of human adipose progenitors into myofibroblasts.The role of primary cilia in obesity and diabetes.Keeping an Eye on Bardet-Biedl Syndrome: A Comprehensive Review of the Role of Bardet-Biedl Syndrome Genes in the Eye.The role of Rpgrip1l, a component of the primary cilium, in adipocyte development and function.The Bardet–Biedl and orofacial digital type 1 ciliopathies
P2860
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P2860
BBS-induced ciliary defect enhances adipogenesis, causing paradoxical higher-insulin sensitivity, glucose usage, and decreased inflammatory response
description
2012 nî lūn-bûn
@nan
2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@ast
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@en
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@en-gb
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@nl
type
label
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@ast
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@en
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@en-gb
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@nl
prefLabel
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@ast
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@en
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@en-gb
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@nl
P2093
P50
P921
P1433
P1476
BBS-induced ciliary defect enh ...... ecreased inflammatory response
@en
P2093
Alban Simon
Anaïs Mockel
Aurélie Claussmann
Catherine Mutter-Schmidt
Cathy Obringer
Charlie De Melo
Corinne Stoetzel
Jean-Philippe Loeffler
Luc Dupuis
Myriam Durand
P304
P356
10.1016/J.CMET.2012.08.005
P407
P577
2012-09-05T00:00:00Z