about
BBS-induced ciliary defect enhances adipogenesis, causing paradoxical higher-insulin sensitivity, glucose usage, and decreased inflammatory responseTransient ciliogenesis involving Bardet-Biedl syndrome proteins is a fundamental characteristic of adipogenic differentiationTrafficking of Endogenous Immunoglobulins by Endothelial Cells at the Blood-Brain Barrier.Bile acids induce energy expenditure by promoting intracellular thyroid hormone activationThe first cellular models based on frataxin missense mutations that reproduce spontaneously the defects associated with Friedreich ataxiaAAV-mediated intramuscular delivery of myotubularin corrects the myotubular myopathy phenotype in targeted murine muscle and suggests a function in plasma membrane homeostasisTemporally controlled targeted somatic mutagenesis in embryonic surface ectoderm and fetal epidermal keratinocytes unveils two distinct developmental functions of BRG1 in limb morphogenesis and skin barrier formationThe prokineticin receptor-1 (GPR73) promotes cardiomyocyte survival and angiogenesisLack of myotubularin (MTM1) leads to muscle hypotrophy through unbalanced regulation of the autophagy and ubiquitin-proteasome pathwaysOral-facial-digital type I protein is required for primary cilia formation and left-right axis specificationThe lipid phosphatase myotubularin is essential for skeletal muscle maintenance but not for myogenesis in miceDual role of COUP-TF-interacting protein 2 in epidermal homeostasis and permeability barrier formationSite-specific Mtm1 mutagenesis by an AAV-Cre vector reveals that myotubularin is essential in adult muscle
P50
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P50
description
hulumtuese
@sq
onderzoeker
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researcher
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հետազոտող
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name
Nadia Messaddeq
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Nadia Messaddeq
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Nadia Messaddeq
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Nadia Messaddeq
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Nadia Messaddeq
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type
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Nadia Messaddeq
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Nadia Messaddeq
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Nadia Messaddeq
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Nadia Messaddeq
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Nadia Messaddeq
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Nadia Messaddeq
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Nadia Messaddeq
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Nadia Messaddeq
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Nadia Messaddeq
@nl
Nadia Messaddeq
@sl