Functional analysis of bone morphogenetic protein type II receptor mutations underlying primary pulmonary hypertension
about
BMP4 induces HO-1 via a Smad-independent, p38MAPK-dependent pathway in pulmonary artery myocytesMolecular pathogenesis of pulmonary arterial hypertensionPrinciples of targeting endothelial cell metabolism to treat angiogenesis and endothelial cell dysfunction in diseaseThe molecular genetics and cellular mechanisms underlying pulmonary arterial hypertensionPulmonary vascular disease related to hemodynamic stress in the pulmonary circulation.Novel signaling pathways in pulmonary arterial hypertension (2015 Grover Conference Series)Delineation of the clinical, molecular and cellular aspects of novel JAM3 mutations underlying the autosomal recessive hemorrhagic destruction of the brain, subependymal calcification, and congenital cataractsAbnormal trafficking of endogenously expressed BMPR2 mutant allelic products in patients with heritable pulmonary arterial hypertensionBone morphogenetic protein 4 enhances canonical transient receptor potential expression, store-operated Ca2+ entry, and basal [Ca2+]i in rat distal pulmonary arterial smooth muscle cellsDownregulation of type II bone morphogenetic protein receptor in hypoxic pulmonary hypertensionDependence of Golgi apparatus integrity on nitric oxide in vascular cells: implications in pulmonary arterial hypertension.Endoglin-mediated suppression of prostate cancer invasion is regulated by activin and bone morphogenetic protein type II receptors.CLAVATA1 dominant-negative alleles reveal functional overlap between multiple receptor kinases that regulate meristem and organ development.Molecular and functional characterization of the BMPR2 gene in Pulmonary Arterial HypertensionSubcellular mechanisms in pulmonary arterial hypertension: combinatorial modalities that inhibit anterograde trafficking and cause bone morphogenetic protein receptor type 2 mislocalization.Heritable forms of pulmonary arterial hypertension.Role of BMPR2 alternative splicing in heritable pulmonary arterial hypertension penetrance.Absence of influence of gender and BMPR2 mutation type on clinical phenotypes of pulmonary arterial hypertension.Transforming growth factor-β inhibition attenuates pulmonary arterial hypertension in rats.Novel promoter and exon mutations of the BMPR2 gene in Chinese patients with pulmonary arterial hypertension.Bone morphogenetic protein 4: potential regulator of shear stress-induced graft neointimal atrophy.Deletion of the sequence encoding the tail domain of the bone morphogenetic protein type 2 receptor reveals a bone morphogenetic protein 7-specific gain of functionPhysiologic and molecular consequences of endothelial Bmpr2 mutation.Protein trafficking dysfunctions: Role in the pathogenesis of pulmonary arterial hypertension.BMP4 increases the expression of TRPC and basal [Ca2+]i via the p38MAPK and ERK1/2 pathways independent of BMPRII in PASMCsFrom developmental disorder to heritable cancer: it's all in the BMP/TGF-beta family.The genetics of pulmonary arterial hypertension in the post-BMPR2 era.Activation of K+ channels: an essential pathway in programmed cell death.Endothelin-1, the unfolded protein response, and persistent inflammation: role of pulmonary artery smooth muscle cells.BMPR2 expression is suppressed by signaling through the estrogen receptor.Metabolomic analysis of bone morphogenetic protein receptor type 2 mutations in human pulmonary endothelium reveals widespread metabolic reprogramming.Activation of the unfolded protein response is associated with pulmonary hypertension.Role of endothelin in fibrosis and anti-fibrotic potential of bosentan.Identification of Biomarkers for Schistosoma-Associated Pulmonary Arterial Hypertension Based on RNA-Seq Data of Mouse Whole Lung Tissues.Pathogenic mechanisms of pulmonary arterial hypertension.Mediators and modulators of pulmonary arterial hypertension.Raf/ERK drives the proliferative and invasive phenotype of BMPR2-silenced pulmonary artery endothelial cells.Mutations of TGFbeta signaling molecules in human disease.Hereditary spastic paraplegia-causing mutations in atlastin-1 interfere with BMPRII trafficking.Molecular pathogenesis of pulmonary arterial hypertension
P2860
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P2860
Functional analysis of bone morphogenetic protein type II receptor mutations underlying primary pulmonary hypertension
description
2002 nî lūn-bûn
@nan
2002 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Functional analysis of bone mo ...... primary pulmonary hypertension
@ast
Functional analysis of bone mo ...... primary pulmonary hypertension
@en
Functional analysis of bone mo ...... primary pulmonary hypertension
@en-gb
Functional analysis of bone mo ...... primary pulmonary hypertension
@nl
type
label
Functional analysis of bone mo ...... primary pulmonary hypertension
@ast
Functional analysis of bone mo ...... primary pulmonary hypertension
@en
Functional analysis of bone mo ...... primary pulmonary hypertension
@en-gb
Functional analysis of bone mo ...... primary pulmonary hypertension
@nl
prefLabel
Functional analysis of bone mo ...... primary pulmonary hypertension
@ast
Functional analysis of bone mo ...... primary pulmonary hypertension
@en
Functional analysis of bone mo ...... primary pulmonary hypertension
@en-gb
Functional analysis of bone mo ...... primary pulmonary hypertension
@nl
P2093
P50
P921
P356
P1476
Functional analysis of bone mo ...... primary pulmonary hypertension
@en
P2093
Hailan Chen
Julia A Flanagan
Nung Rudarakanchana
Paul D Upton
P304
P356
10.1093/HMG/11.13.1517
P407
P577
2002-06-15T00:00:00Z