Metabolomic analysis of bone morphogenetic protein receptor type 2 mutations in human pulmonary endothelium reveals widespread metabolic reprogramming. - Wikidata - wikimedia - TriplyDB

Metabolomic analysis of bone morphogenetic protein receptor type 2 mutations in human pulmonary endothelium reveals widespread metabolic reprogramming.

Metabolomic analysis of bone morphogenetic protein receptor type 2 mutations in human pulmonary endothelium reveals widespread metabolic reprogramming.

http://www.wikidata.org/entity/Q36108056

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Pulmonary hypertension in patients with heart failure and preserved ejection fraction: differential diagnosis and management Redox biology in pulmonary arterial hypertension (2013 Grover Conference Series)BMPR2 spruces up the endothelium in pulmonary hypertension Principles of targeting endothelial cell metabolism to treat angiogenesis and endothelial cell dysfunction in disease Metabolic dysfunction in pulmonary hypertension: the expanding relevance of the Warburg effect Integration of complex data sources to provide biologic insight into pulmonary vascular disease (2015 Grover Conference Series)Role of oxidized lipids in pulmonary arterial hypertension A Biochemical Approach to Understand the Pathogenesis of Advanced Pulmonary Arterial Hypertension: Metabolomic Profiles of Arginine, Sphingosine-1-Phosphate, and Heme of Human Lung Emerging Metabolic Therapies in Pulmonary Arterial Hypertension.Plasma acylcarnitines are associated with pulmonary hypertension.Dysfunctional BMPR2 signaling drives an abnormal endothelial requirement for glutamine in pulmonary arterial hypertension The emerging role of metabolomics in the development of biomarkers for pulmonary hypertension and other cardiovascular diseases (2013 Grover Conference series)Metabolomic heterogeneity of pulmonary arterial hypertension Mitochondrial dynamics in pulmonary arterial hypertension.Impaired right ventricular hemodynamics indicate preclinical pulmonary hypertension in patients with metabolic syndrome.Rescuing the BMPR2 signaling axis in pulmonary arterial hypertension Increased Mutagen Sensitivity and DNA Damage in Pulmonary Arterial Hypertension.RNA Sequencing Analysis Detection of a Novel Pathway of Endothelial Dysfunction in Pulmonary Arterial Hypertension.Expression of mutant bone morphogenetic protein receptor II worsens pulmonary hypertension secondary to pulmonary fibrosis.Estrogen Metabolite 16α-Hydroxyestrone Exacerbates Bone Morphogenetic Protein Receptor Type II-Associated Pulmonary Arterial Hypertension Through MicroRNA-29-Mediated Modulation of Cellular Metabolism Role of carnitine acetyl transferase in regulation of nitric oxide signaling in pulmonary arterial endothelial cells Metabolic Dysfunction in Pulmonary Arterial Hypertension.Fatty Acid Metabolic Defects and Right Ventricular Lipotoxicity in Human Pulmonary Arterial Hypertension.Vascular stiffness mechanoactivates YAP/TAZ-dependent glutaminolysis to drive pulmonary hypertension Induction of pulmonary hypertensive changes by extracellular vesicles from monocrotaline-treated mice.Hyperoxia synergizes with mutant bone morphogenic protein receptor 2 to cause metabolic stress, oxidant injury, and pulmonary hypertension.Evidence for right ventricular lipotoxicity in heritable pulmonary arterial hypertension.Proteomics of pulmonary hypertension: could personalized profiles lead to personalized medicine?De novo synthesize of bile acids in pulmonary arterial hypertension lung.Fatty acid metabolism in pulmonary arterial hypertension: role in right ventricular dysfunction and hypertrophy.Oestrogen inhibition reverses pulmonary arterial hypertension and associated metabolic defects.Hallmarks of Pulmonary Hypertension: Mesenchymal and Inflammatory Cell Metabolic Reprogramming.Pyridine Dinucleotides from Molecules to Man.Mechanisms of Lipid Accumulation in the Bone Morphogenetic Protein Receptor Type 2 Mutant Right Ventricle.The diversified function and potential therapy of ectopic olfactory receptors in non-olfactory tissues.Involvement of the Warburg effect in non-tumor diseases processes.Limiting Injury During Saphenous Vein Graft Preparation For Coronary Arterial Bypass Prevents Metabolic Decompensation.Identification of miR-124 as a Major Regulator of Enhanced Endothelial Cell Glycolysis in Pulmonary Arterial Hypertension via PTBP1 and PKM2.Pentose Shunt, Glucose-6-Phosphate Dehydrogenase, NADPH Redox, and Stem Cells in Pulmonary Hypertension.Single cell cloning generates lung endothelial colonies with conserved growth, angiogenic, and bioenergetic characteristics.

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P2860

Metabolomic analysis of bone morphogenetic protein receptor type 2 mutations in human pulmonary endothelium reveals widespread metabolic reprogramming.

http://www.wikidata.org/entity/Q36108056

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Metabolomic analysis of bone m ...... pread metabolic reprogramming.

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Metabolomic analysis of bone m ...... pread metabolic reprogramming.

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Metabolomic analysis of bone m ...... pread metabolic reprogramming.

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Metabolomic analysis of bone m ...... pread metabolic reprogramming.

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Anna R Hemnes

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Bryan M Wittmann

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James D West

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Joshua P Fessel

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Koichiro Tatsumi

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Linda J Robinson

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Nobuhiro Tanabe

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Rizwan Hamid

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Tom Blackwell

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Yuji Tada

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P2860

Functional analysis of bone morphogenetic protein type II receptor mutations underlying primary pulmonary hypertension

Understanding the Warburg effect: the metabolic requirements of cell proliferation

IDH1 and IDH2 mutations in gliomas

Metabolomic profiles delineate potential role for sarcosine in prostate cancer progression

Direct signaling by the BMP type II receptor via the cytoskeletal regulator LIMK1

Statistical significance for genomewide studies

Organization of GC/MS and LC/MS metabolomics data into chemical libraries

An abnormal mitochondrial-hypoxia inducible factor-1alpha-Kv channel pathway disrupts oxygen sensing and triggers pulmonary arterial hypertension in fawn hooded rats: similarities to human pulmonary arterial hypertension

Leukemic IDH1 and IDH2 mutations result in a hypermethylation phenotype, disrupt TET2 function, and impair hematopoietic differentiation

Dichloroacetate prevents and reverses pulmonary hypertension by inducing pulmonary artery smooth muscle cell apoptosis.

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