Evidence that BCL-2 represses apoptosis by regulating endoplasmic reticulum-associated Ca2+ fluxes
about
Calcium regulation of Ndr protein kinase mediated by S100 calcium-binding proteinsActivation of K+ channels induces apoptosis in vascular smooth muscle cellsCharacterization of fortilin, a novel antiapoptotic proteinp28 Bap31, a Bcl-2/Bcl-XL- and procaspase-8-associated protein in the endoplasmic reticulumConformation of the Bax C-terminus regulates subcellular location and cell deathCACNA2D2-mediated apoptosis in NSCLC cells is associated with alterations of the intracellular calcium signaling and disruption of mitochondria membrane integrityNAIP interacts with hippocalcin and protects neurons against calcium-induced cell death through caspase-3-dependent and -independent pathwaysCa(2+) transfer from the ER to mitochondria: when, how and whyCalcineurin functions in Ca(2+)-activated cell death in mammalian cellsDexamethasone protected human glioblastoma U87MG cells from temozolomide induced apoptosis by maintaining Bax:Bcl-2 ratio and preventing proteolytic activitiesTargeting the Bcl-2 family for cancer therapyMISC-1/OGC links mitochondrial metabolism, apoptosis and insulin secretionER Stress-Mediated Signaling: Action Potential and Ca(2+) as Key PlayersThe cell death-promoting gene DP5, which interacts with the BCL2 family, is induced during neuronal apoptosis following exposure to amyloid beta proteinCritical upstream signals of cytochrome C release induced by a novel Bcl-2 inhibitorCloning of AIP1, a novel protein that associates with the apoptosis-linked gene ALG-2 in a Ca2+-dependent reactionThe Ca2+ concentration of the endoplasmic reticulum is a key determinant of ceramide-induced apoptosis: significance for the molecular mechanism of Bcl-2 actionResponse of neurons to an irreversible inhibition of endoplasmic reticulum Ca(2+)-ATPase: relationship between global protein synthesis and expression and translation of individual genesThe copper chelator ATN-224 induces peroxynitrite-dependent cell death in hematological malignanciesMitochondrial adaptations to oxidative stress confer resistance to apoptosis in lymphoma cellsFactors determining sensitivity and resistance of tumor cells to arsenic trioxideCHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulumTumor necrosis factor induces apoptosis in hepatoma cells by increasing Ca(2+) release from the endoplasmic reticulum and suppressing Bcl-2 expressionPhosphorylation of BCL-2 regulates ER Ca2+ homeostasis and apoptosis.Mitochondrial dysfunction in bipolar disorder.Changes in phosphate metabolism in thymoma cells suggest mechanisms for resistance to dexamethasone-induced apoptosis. A 31P NMR spectroscopic study of cell extracts.Neuronal apoptosis induced by pharmacological concentrations of 3-hydroxykynurenine: characterization and protection by dantrolene and Bcl-2 overexpression.The apoptosis promoting Bcl-2 homologues Bak and Nbk/Bik overcome drug resistance in Mdr-1-negative and Mdr-1-overexpressing breast cancer cell lines.The phosphorylation status and anti-apoptotic activity of Bcl-2 are regulated by ERK and protein phosphatase 2A on the mitochondria.Calcium signaling and cytotoxicity.Role of the Bcl-2 gene family in prostate cancer progression and its implications for therapeutic intervention.Sigma-1 receptors regulate Bcl-2 expression by reactive oxygen species-dependent transcriptional regulation of nuclear factor kappaBThe dual role of calcium as messenger and stressor in cell damage, death, and survival.Developmental transcriptome profiling of bovine muscle tissue reveals an abundant GosB that regulates myoblast proliferation and apoptosis.Long term lithium treatment suppresses p53 and Bax expression but increases Bcl-2 expression. A prominent role in neuroprotection against excitotoxicity.The characteristics of Bax inhibitor-1 and its related diseases.Downregulation of Smac attenuates H2O2-induced apoptosis via endoplasmic reticulum stress in human lens epithelial cellsBcl-2 and Bcl-X(L) block thapsigargin-induced nitric oxide generation, c-Jun NH(2)-terminal kinase activity, and apoptosisConcerted regulation of wild-type p53 nuclear accumulation and activation by S100B and calcium-dependent protein kinase C.Angiogenin prevents serum withdrawal-induced apoptosis of P19 embryonal carcinoma cells
P2860
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P2860
Evidence that BCL-2 represses apoptosis by regulating endoplasmic reticulum-associated Ca2+ fluxes
description
1994 nî lūn-bûn
@nan
1994 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
name
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@ast
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@en
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@en-gb
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@nl
type
label
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@ast
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@en
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@en-gb
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@nl
prefLabel
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@ast
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@en
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@en-gb
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@nl
P2093
P2860
P921
P3181
P356
P1476
Evidence that BCL-2 represses ...... ticulum-associated Ca2+ fluxes
@en
P2093
P2860
P304
P3181
P356
10.1073/PNAS.91.14.6569
P407
P577
1994-07-01T00:00:00Z