CD26 mediates dissociation of Tollip and IRAK-1 from caveolin-1 and induces upregulation of CD86 on antigen-presenting cells - Wikidata - wikimedia - TriplyDB

CD26 mediates dissociation of Tollip and IRAK-1 from caveolin-1 and induces upregulation of CD86 on antigen-presenting cells

CD26 mediates dissociation of Tollip and IRAK-1 from caveolin-1 and induces upregulation of CD86 on antigen-presenting cells

http://www.wikidata.org/entity/Q24529963

about

Caveolin-1 mediates Fas-BID signaling in hyperoxia-induced apoptosis Caveolin-1 triggers T-cell activation via CD26 in association with CARMA1 Recent Advances in Dipeptidyl-Peptidase-4 Inhibition Therapy: Lessons from the Bench and Clinical Trials The mechanism of excessive intestinal inflammation in necrotizing enterocolitis: an immature innate immune response.Soluble CD26/Dipeptidyl Peptidase IV Enhances the Transcription of IL-6 and TNF-α in THP-1 Cells and Monocytes.Probiotics prevent necrotizing enterocolitis by modulating enterocyte genes that regulate innate immune-mediated inflammation.Differential regulation of Foxp3 and IL-17 expression in CD4 T helper cells by IRAK-1 The interleukin-1 receptor associated kinase 1 contributes to the regulation of NFAT The involvement of the interleukin-1 receptor-associated kinases (IRAKs) in cellular signaling networks controlling inflammation Caveolin-1 Tyr14 phosphorylation induces interaction with TLR4 in endothelial cells and mediates MyD88-dependent signaling and sepsis-induced lung inflammation.The dipeptidyl peptidase IV family in cancer and cell biology.Caveolin-1: a critical regulator of lung injury.DPP4 in cardiometabolic disease: recent insights from the laboratory and clinical trials of DPP4 inhibition.Myocardial tissue caveolae.DPP-4 (CD26) inhibitor sitagliptin exerts anti-inflammatory effects on rat insulinoma (RINm) cells via suppressing NF-κB activation.DPP-IV inhibitor anagliptin exerts anti-inflammatory effects on macrophages, adipocytes, and mouse livers by suppressing NF-κB activation.CD26 and Asthma: a Comprehensive Review.Loss of the innate immunity negative regulator IRAK-M leads to enhanced host immune defense against tumor growth.Tollip-induced down-regulation of MARCH1.CD26-mediated co-stimulation in human CD8(+) T cells provokes effector function via pro-inflammatory cytokine production.Identification of senescent cell surface targetable protein DPP4.Higher serum DPP-4 enzyme activity and decreased lymphocyte CD26 expression in type 1 diabetes.Hepatocyte-secreted DPP4 in obesity promotes adipose inflammation and insulin resistance.

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P2860

CD26 mediates dissociation of Tollip and IRAK-1 from caveolin-1 and induces upregulation of CD86 on antigen-presenting cells

http://www.wikidata.org/entity/Q24529963

description

2005 nî lūn-bûn

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2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

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CD26 mediates dissociation of ...... 86 on antigen-presenting cells

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CD26 mediates dissociation of ...... 86 on antigen-presenting cells

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CD26 mediates dissociation of ...... 86 on antigen-presenting cells

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P356

MCB.25.17.7743-7757.2005

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Molecular and Cellular Biology

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CD26 mediates dissociation of ...... 86 on antigen-presenting cells

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Chikao Morimoto

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Hiroshi Kawasaki

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Hirotoshi Tanaka

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Kei Ohnuma

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Kunika Nishibashi

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Masahiko Uchiyama

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Nam H Dang

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Osamu Hosono

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Satoshi Iwata

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Tadanori Yamochi

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P2860

Tollip, a new component of the IL-1RI pathway, links IRAK to the IL-1 receptor

Internalization of CD26 by mannose 6-phosphate/insulin-like growth factor II receptor contributes to T cell activation

IRAK-M is a negative regulator of Toll-like receptor signaling

IRAK: a kinase associated with the interleukin-1 receptor

A ubiquitin-binding motif required for intramolecular monoubiquitylation, the CUE domain

Impaired cytokine signaling in mice lacking the IL-1 receptor-associated kinase

Caveolin-1 associates with TRAF2 to form a complex that is recruited to tumor necrosis factor receptors

Translocation of the interleukin-1 receptor-associated kinase-1 (IRAK-1) into the nucleus

Decreased expression of Toll-like receptor-4 and MD-2 correlates with intestinal epithelial cell protection against dysregulated proinflammatory gene expression in response to bacterial lipopolysaccharide

Negative regulation of toll-like receptor-mediated signaling by Tollip

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10.1128/MCB.25.17.7743-7757.2005

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