SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness - Wikidata - wikimedia - TriplyDB

SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

http://www.wikidata.org/entity/Q24530758

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Inhibition of mutation and combating the evolution of antibiotic resistance Antibiotics and UV radiation induce competence for natural transformation in Legionella pneumophila Insights into the complex levels of regulation imposed on Escherichia coli DNA polymerase V Culture history and population heterogeneity as determinants of bacterial adaptation: the adaptomics of a single environmental transition Ribosomal frameshifting and transcriptional slippage: From genetic steganography and cryptography to adventitious use Structural Insight into Translesion Synthesis by DNA Pol II A strategically located serine residue is critical for the mutator activity of DNA polymerase IV from Escherichia coli DNA polymerase II as a fidelity factor in chromosomal DNA synthesis in Escherichia coli Diversification rates increase with population size and resource concentration in an unstructured habitat.Acinetobacter baylyi starvation-induced genes identified through incubation in long-term stationary phase.Optimization of DNA polymerase mutation rates during bacterial evolution.Error-prone DNA polymerase IV is regulated by the heat shock chaperone GroE in Escherichia coli Role of the DinB homologs Rv1537 and Rv3056 in Mycobacterium tuberculosis Error-prone DNA polymerase IV is controlled by the stress-response sigma factor, RpoS, in Escherichia coli.Characterization of three mycobacterial DinB (DNA polymerase IV) paralogs highlights DinB2 as naturally adept at ribonucleotide incorporation.The Thermotoga maritima phenotype is impacted by syntrophic interaction with Methanococcus jannaschii in hyperthermophilic coculture Translesion DNA polymerases Polynucleobacter necessarius, a model for genome reduction in both free-living and symbiotic bacteria.Translesion DNA synthesis and mutagenesis in prokaryotes.Mycobacterium smegmatis DinB2 misincorporates deoxyribonucleotides and ribonucleotides during templated synthesis and lesion bypass The SMC-like protein complex SbcCD enhances DNA polymerase IV-dependent spontaneous mutation in Escherichia coli Translesion DNA Synthesis.General stress response regulator RpoS in adaptive mutation and amplification in Escherichia coli.Aeons of distress: an evolutionary perspective on the bacterial SOS response.Tuberculosis chemotherapy: the influence of bacillary stress and damage response pathways on drug efficacy.Role of DNA polymerase IV in Escherichia coli SOS mutator activity Role of high-fidelity Escherichia coli DNA polymerase I in replication bypass of a deoxyadenosine DNA-peptide cross-link Stationary phase mutagenesis: mechanisms that accelerate adaptation of microbial populations under environmental stress.Urinary tract infection drives genome instability in uropathogenic Escherichia coli and necessitates translesion synthesis DNA polymerase IV for virulence Double-Strand Break Repair and Holliday Junction Processing Are Required for Chromosome Processing in Stationary-Phase Escherichia coli Cells Targeted gene evolution in Escherichia coli using a highly error-prone DNA polymerase I.MsDpo4-a DinB Homolog from Mycobacterium smegmatis-Is an Error-Prone DNA Polymerase That Can Promote G:T and T:G Mismatches.Mutation as a stress response and the regulation of evolvability Error-prone DNA repair system in enteroaggregative Escherichia coli identified by subtractive hybridization.6S RNA function enhances long-term cell survival.Interactions and Localization of Escherichia coli Error-Prone DNA Polymerase IV after DNA Damage.Stress responses and genetic variation in bacteria.DNA replication fidelity in Escherichia coli: a multi-DNA polymerase affair.Role of accessory DNA polymerases in DNA replication in Escherichia coli: analysis of the dnaX36 mutator mutant.Bacterial stationary-state mutagenesis and Mammalian tumorigenesis as stress-induced cellular adaptations and the role of epigenetics.

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P2860

SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

http://www.wikidata.org/entity/Q24530758

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2002 nî lūn-bûn

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2002 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2002 թվականի հունիսին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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type

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

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Proceedings of the National Academy of Sciences of the United States of America

P1476

SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

@en

P2093

Bethany Yeiser

http://www.w3.org/2001/XMLSchema#string

Evan D Pepper

http://www.w3.org/2001/XMLSchema#string

Myron F Goodman

http://www.w3.org/2001/XMLSchema#string

Steven E Finkel

http://www.w3.org/2001/XMLSchema#string

P2860

Adaptive mutation: the uses of adversity

All three SOS-inducible DNA polymerases (Pol II, Pol IV and Pol V) are involved in induced mutagenesis

Multiple pathways for SOS-induced mutagenesis in Escherichia coli: an overexpression of dinB/dinP results in strongly enhancing mutagenesis in the absence of any exogenous treatment to damage DNA

Roles of DNA polymerases V and II in SOS-induced error-prone and error-free repair in Escherichia coli

DNA polymerase II

The importance of repairing stalled replication forks

The Y-family of DNA polymerases

The mutagenesis protein UmuC is a DNA polymerase activated by UmuD', RecA, and SSB and is specialized for translesion replication.

Mismatch repair is diminished during stationary-phase mutation.

DNA polymerase II is encoded by the DNA damage-inducible dinA gene of Escherichia coli.

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8737-8741

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scholarly article

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4497011

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10.1073/PNAS.092269199

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13

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99

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2002-06-11T00:00:00Z

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11313095

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12060704

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124368

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